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Slow-release carbohydrates: growing evidence on metabolic responses and public health interest. Summary of the symposium held at the 12th European Nutrition Conference (FENS 2015)

机译:缓慢释放的碳水化合物:关于代谢反应和公共卫生利益的越来越多的证据。在第十二届欧洲营养大会上举行的专题讨论会摘要(FENS 2015)

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摘要

To draw attention to the necessity of considering differences in the digestibility of carbohydrates, and more specifically of starch, a symposium was held at the 12th European Nutrition Conference (FENS), which took place in Berlin from October 20 to 23, 2015. The purpose of this session was to present the consolidated knowledge and recent advances regarding the relationship between slow-release carbohydrates, metabolic responses, and public health issues. Three main topics were presented: 1) the definition of, sources of, and recognised interest in the glycaemic response to slowly digestible starch (SDS); 2) clinical evidence regarding the physiological effects of slow-release carbohydrates from cereal foods; and 3) interest in reducing the postprandial glycaemic response to help prevent metabolic diseases. Foods with the highest SDS content induce the lowest glycaemic responses, as the starch is protected from gelatinisation during processing. In humans, high-SDS food consumption induces slower glucose release, lower postprandial insulinaemia, and stimulation of gut hormones. Moreover, postprandial hyperglycaemia is an independent risk factor for type two diabetes mellitus (T2DM) and cardiovascular disease (CVD). Therefore, given the plausible aetiologic mechanisms, we argue that postprandial glucose levels are relevant for health and disease and represent a meaningful target for intervention, for example, through dietary factors. This symposium was organised by Mondelez International R&D.
机译:为提请注意考虑碳水化合物(尤其是淀粉)的消化率差异的必要性,于2015年10月20日至23日在柏林举行的第十二届欧洲营养会议(FENS)上举行了一次专题讨论会。本次会议的目的是介绍有关缓释碳水化合物,代谢反应和公共卫生问题之间关系的综合知识和最新进展。提出了三个主要主题:1)对慢消化淀粉(SDS)的血糖反应的定义,来源和公认的兴趣; 2)关于谷物食品中缓释碳水化合物的生理效应的临床证据; 3)降低餐后血糖反应以预防代谢性疾病的兴趣。 SDS含量最高的食品引起的血糖反应最低,这是因为淀粉在加工过程中可以防止糊化。在人类中,高SDS的食物摄入量会导致葡萄糖释放缓慢,餐后胰岛素血症降低以及肠激素刺激。此外,餐后高血糖是2型糖尿病(T2DM)和心血管疾病(CVD)的独立危险因素。因此,鉴于合理的病因学机制,我们认为餐后血糖水平与健康和疾病相关,并且代表通过饮食因素进行干预的有意义的目标。这次研讨会是由Mondelez International R&D组织的。

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