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Exendin-4 Inhibits the Expression of SEPP1 and Fetuin-A via Improvement of Palmitic Acid-Induced Endoplasmic Reticulum Stress by AMPK

机译：Exendin-4通过改善AMPK诱导的棕榈酸诱导的内质网应激来抑制SEPP1和Fetuin-A的表达

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摘要

BackgroundSelenoprotein P (SEPP1) and fetuin-A, both circulating liver-derived glycoproteins, are novel biomarkers for insulin resistance and nonalcoholic fatty liver disease. However, the effect of exendin-4 (Ex-4), a glucagon-like peptide-1 receptor agonist, on the expression of hepatokines, SEPP1, and fetuin-A, is unknown.

机译：背景硒蛋白P（SEPP1）和胎球蛋白-A，都是循环的肝糖蛋白，是胰岛素抵抗和非酒精性脂肪肝疾病的新型生物标志物。但是，未知胰高血糖素样肽-1受体激动剂exendin-4（Ex-4）对肝因子，SEPP1和胎球蛋白A的表达的影响。

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期刊名称 Endocrinology and Metabolism
作者
Jinmi Lee; Seok-Woo Hong; Se Eun Park; Eun-Jung Rhee; Cheol-Young Park; Ki-Won Oh; Sung-Woo Park; Won-Young Lee;
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作者单位

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年(卷),期 2015(30),2
年度 2015
页码 177–184
总页数 8
原文格式 PDF
正文语种
中图分类基础医学;
关键词
Exendin-4 Palmitic acid Endoplasmic reticulum stress AMP-activated protein kinases Selenoprotein P Fetuin-A Hepatokine;

机译：Exendin-4;棕榈酸;内质网应激;AMP激活的蛋白激酶;Selenoprotein P;Fetuin-A;肝素;

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专利

1. Kaempferol alleviates palmitic acid-induced lipid stores, endoplasmic reticulum stress and pancreatic beta-cell dysfunction through AMPK/mTOR-mediated lipophagy [J] . Varshney Ritu, Varshney Rajat, Mishra Rutusmita, The Journal of Nutritional Biochemistry . 2018,第期

机译：Kaempferol通过AMPK / MTOR介导的PIPOPHAGY减轻棕榈酸诱导的棕榈酸诱导的脂质储存，内质网胁迫和胰腺β细胞功能障碍
2. Curcumin attenuates palmitic acid-induced cell apoptosis by inhibiting endoplasmic reticulum stress in H9C2 cardiomyocytes [J] . Guan G., Lei L., Lv Q., Human and Experimental Toxicology . 2019,第6期

机译：姜黄素通过抑制H9C2心肌细胞中的内质网胁迫抑制棕榈酸诱导的细胞凋亡
3. Role of hepatocyte S6K1 in palmitic acid-induced endoplasmic reticulum stress, lipotoxicity, insulin resistance and in oleic acid-induced protection [J] . Pardo Virginia, Gonzalez-Rodriguez Agueda, Muntane Jordi, Food and Chemical Toxicology: An International Journal Published for the British Industrial Biological Research . 2015,第Null期

机译：肝细胞S6K1在棕榈酸诱导的内质网应激，脂毒性，胰岛素抵抗以及油酸诱导的保护中的作用
4. Fragment-based drug design of host endoplasmic reticulum α-glucosidase II inhibitors for dengue fever treatment using an integrated computational approach [C] . E. P. Toepak, M. A. F. Nasution, U. S. F. Tambunan International Symposium on Current Progress in Mathematics and Sciences . 2018

机译：基于片段的宿主内质网的药物设计α-葡萄糖蛋白II抑制剂，用于使用综合计算方法进行登革热治疗
5. Potential role of endoplasmic reticulum redox changes in endoplasmic reticulum stress and impaired protein folding in obesity-associated insulin resistance [D] . Sarkar, Deboleena 2013

机译：内质网氧化还原变化在内质网应激和蛋白质折叠受损在肥胖相关胰岛素抵抗中的潜在作用
6. Exendin-4 a glucagon-like peptide-1 receptor agonist inhibits Aβ25-35-induced apoptosis in PC12 cells by suppressing the expression of endoplasmic reticulum stress-related proteins [O] . Jianfeng Zhang, Junfeng Wu, Weichen Zeng, 2015

机译：胰高血糖素样肽1受体激动剂Exendin-4通过抑制内质网应激相关蛋白的表达来抑制Aβ25-35诱导的PC12细胞凋亡。
7. Exendin-4 Inhibits the Expression of SEPP1 and Fetuin-A via Improvement of Palmitic Acid-Induced Endoplasmic Reticulum Stress by AMPK [O] . Jinmi Lee, Seok-Woo Hong, Se Eun Park, 2015

机译：Exendin-4通过ampK改善棕榈酸诱导的内质网应激抑制sEpp1和胎球蛋白-a的表达

Exendin-4 Inhibits the Expression of SEPP1 and Fetuin-A via Improvement of Palmitic Acid-Induced Endoplasmic Reticulum Stress by AMPK

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