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Natural history of Pott’s kyphosis

机译:波特后凸症的自然史

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摘要

The natural history of Pott’s kyphosis is different from that of other spinal deformities. After healing of the spinal infection, the post-tubercular kyphosis in adults is static but in children variable progression of the kyphosis is seen. The changes occurring in the spine of children, after the healing of the tubercular lesion, are more significant than the changes that occur during the active stage of infection. During growth, there is a decrease in deformity in 44 % of the children, an increase in deformity in 39 % of the children and no change in deformity in 17 % of the children. The critical factor leading to the progress of the deformity is dislocation of the facets. This can be identified on radiographs by the “Spine-at-risk” signs. Dislocation of facets at more than two levels can lead to the “Buckling collapse” of the spine, which is characteristically seen only in severe tubercular kyphosis in children. Age below 10 years, vertebral body loss of more than 1–1.5 pre-treatment deformity angle of greater than 30° and involvement of cervicothoracic or thoracolumbar junction are the other risk factors for deformity progression. In children, the kyphosis can progress even after healing of the spinal infection and hence children with spinal tuberculosis must be followed-up till skeletal maturity.
机译:Pott的后凸畸形的自然史与其他脊柱畸形的自然史不同。脊柱感染治愈后,成人的结核后后凸是静止的,但在儿童中则可看到后凸的变化。结核病灶治愈后,儿童脊柱发生的变化比感染活跃期发生的变化更为显着。在成长过程中,有44%的儿童畸形减少了,有39%的儿童畸形增加了,有17%的儿童畸形没有变化。导致畸形发展的关键因素是小平面的错位。可以在放射线照片上通过“脊柱有危险”的标志来识别。小平面的错位超过两个级别会导致脊椎“屈曲塌陷”,这通常仅在儿童的严重结核性后凸畸形中可见。年龄在10岁以下,椎体损失大于1–1.5,大于30°的治疗前畸形角以及累及子宫颈胸腔或胸腰椎交界处是导致畸形进展的其他危险因素。在儿童中,即使在脊柱感染治愈后,后凸也会发展,因此必须对患有脊椎结核的儿童进行随访直至骨骼成熟。

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