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刘小伟; 游宇; 等;
中南大学湘雅二医院消化科410011;
LPS; 信号传导分子; TLR4; 小鼠; 肝损伤; 研究;
机译:4-羟基苯苯并[D]恶唑-2(3H) - 通过抑制小鼠的TLR4 / NF-Kappa B和Mapk信号传导途径来改善LPS / D-Galn诱导的急性肝损伤
机译:通过调节LPS / TLR4信号传导途径,石奈甙通过调节LPS / TLR4信号传导途径引起的高脂肪饮食诱导的非酒精性脂肪肝疾病
机译:在衰老期间,野生型小鼠对LPS的小胶质细胞反应增加,但在阿尔茨海默氏症小鼠模型中减弱:TLR4信号传导在疾病进展中的意义
机译:VIP降低了LPS和TNF-A治疗在人类滑膜成纤维细胞中的诱导的TLR4表达
机译:PTP alpha缺陷小鼠的分子信号传导缺陷研究:胰岛素和NMDA受体信号传导。
机译:基因组范围表达谱分析和诱变研究表明Lps响应似乎是完全依赖于TLR4和mD-2的表达并取决于分子间的离子相互作用
机译:Decursin负调节LPS诱导的TLR4和JNK信号传导通过PRP4体外表达刺激的TLR4和JNK信号传导
机译:Lps-低反应性和Lps反应小鼠对革兰氏阴性菌鼠伤寒沙门氏菌和肺炎克雷伯菌的炎症反应
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机译:TLR4 LPS通过与LPS的TLR4结合竞争而具有抗炎和抗氧化活性的新化合物及其医学用途
机译:方法在合成和合成pia-alpha-alpha-alpha-alpha-alpha-alpha-alpha 同时,有必要研究两者之间的关系。
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