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Experimental and theoretical assessment of flow asymmetries in normal and pathological speech.

机译:正常和病理性语音中流量不对称的实验和理论评估。

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摘要

Speech is initiated when a critical pressure is achieved in the lungs, forcing the vocal folds apart. As the air is pushed up the trachea and through the larynx, the resulting aerodynamic forces, coupled with the vocal fold tissue properties, excite self-sustained oscillations of the vocal folds, which form the basis for vocalized speech. Voiced speech is a complex process which involves the inter-dependency of fluid flow, tissue properties, and acoustics. The speech process becomes even more complicated by the introduction of neurogenic and structural pathologies which may disrupt vocal fold motion and alter fluid behavior and/or the acoustical response. Recurrent laryngeal nerve paralysis is the most common neurogenic pathology resulting from damage to the vagus nerve which innervates all of the muscles of the larynx except the cricothyroid. Recurrent laryngeal nerve paralysis usually results in the complete immobility of the damaged vocal fold. Unilateral polyps, characterized by large growths on the medial surface of the vocal folds, are a common structural pathology that most often results from misuse and abuse of the voice.;Flow through 7.5 times scaled-up driven vocal fold models in a pressure-driven flow facility was investigated for both normal and pathological speech conditions over a range of physiologically relevant flow rates. The glottal jet trajectory was resolved during the divergent portions of the phonatory cycle. The glottal jet assumed a bi-modal trajectory for normal speech. Vocal fold paresis and paralysis were modeled by limiting the motion of one vocal fold wall, which resulted in a stable, stationary attachment of the flow to the impaired vocal fold wall. The presence of a unilateral polyp introduced (1) large spatial variations in the flow in both the anterior-posterior and the inferior-superior directions and (2) fluctuations in the flow separation points that were atypical compared to those found during normal vocal fold motion.;A theoretical solution for flow over an infinite flat plate that is translating and rotating at constant velocity in a direction normal to the freestream velocity was developed for application to intraglottal flows. The solution addresses the role of boundary conditions on flow stability. It is shown that downstream of the point of rotation, the influence of a rotating boundary acts as a favorable pressure gradient, stabilizing the flow field. A theoretical self-similarity solution was derived in the rotating reference frame for flow over the rotating and translating flat plate, allowing direct application of the solution to flow within the glottis. Reasonable agreement (∼ 5%) between the theory and the experimental results was found. A new procedure for applying the theoretical solution to multi-mass models of speech is proposed, with comparison to the often-employed, but inappropriate, Bernoulli flow assumption.
机译:当肺部达到临界压力,迫使声带分开时,就会启动语音。当空气被推上气管并通过喉部时,所产生的空气动力与声带的组织特性相结合,激发了声带的自持振荡,这构成了发声的基础。语音语音是一个复杂的过程,涉及到流体流动,组织特性和声学之间的相互依赖性。通过引入神经源性和结构性病理学,语音过程变得更加复杂,这可能会破坏声带运动并改变体液行为和/或声学反应。喉返神经麻痹是最常见的神经源性病理,是由于迷走神经受损所致,迷走神经支配除环甲亢以外的喉部所有肌肉。喉返神经麻痹通常导致受损的声带完全不活动。单侧息肉的特征是声带内侧表面大量生长,是常见的结构病理,最常见的原因是误用和滥用声音;在压力驱动下流经7.5倍放大驱动的声带模型在生理相关流量范围内,对正常和病理性言语条件下的流量设备进行了研究。在发声周期的发散部分期间,声门喷射轨迹得以解决。声门喷射假设正常语音为双峰轨迹。通过限制一个声带壁的运动来模拟声带麻痹和瘫痪,从而使血流稳定,固定地附着在受损的声带壁上。单侧息肉的存在导致(1)前后方向和上下方向的血流都有较大的空间变化,以及(2)与正常人声带运动相比不典型的血流分离点波动提出了一种理论上的解决方案,用于在无限平板上流动,该平板在与自由流速度垂直的方向上以恒定速度平移和旋转,并应用于声门内流动。该解决方案解决了边界条件对流动稳定性的作用。结果表明,在旋转点的下游,旋转边界的影响起到了有利的压力梯度的作用,从而稳定了流场。在旋转参考系中导出了理论上的自相似解,以使之流过旋转和平移的平板,从而允许将溶液直接应用到声门内。在理论和实验结果之间找到了合理的一致性(〜5%)。与经常使用但不合适的伯努利流量假设相比较,提出了一种将理论解应用于多质量语音模型的新方法。

著录项

  • 作者

    Erath, Byron D.;

  • 作者单位

    Purdue University.;

  • 授予单位 Purdue University.;
  • 学科 Health Sciences Speech Pathology.;Engineering Mechanical.;Engineering Biomedical.
  • 学位 Ph.D.
  • 年度 2009
  • 页码 314 p.
  • 总页数 314
  • 原文格式 PDF
  • 正文语种 eng
  • 中图分类
  • 关键词

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