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Influence of DISH, Ankylosis, Spondylosis and Osteophytes on Serious-to-Fatal Spinal Fractures and Cord Injury in Rear Impacts

机译:培养皿,触角,脊柱病和骨折对致命对致命脊髓裂缝和脐带损伤的影响

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Seats have become stronger over the past two decades and remain more upright in rear impacts. While head restraints are higher and more forward providing support for the head and neck, serious-to-fatal injuries to the thoracic and cervical spine have been seen in occupants with spinal disorders, such as DISH (diffuse idiopathic skeletal hyperostosis), ankylosis, spondylosis and/or osteophytes that ossify the joints in the spine. This case study addresses the influence of spinal disorders on fracture-dislocation and spinal cord injury in rear impacts with relatively upright seats. Nineteen field accidents were investigated where serious-to-fatal injuries of the thoracic and cervical spine occurred with the seat remaining upright or slightly reclined. The occupants were lap-shoulder belted, some with belt pretensioning and cinching latch plate. The occupants were older and had pre-existing disorders of the spine, including DISH, ankylosis, spondylosis and/or osteophytes that ossify the spinal joints. The crashes were summarized and the mechanism for injury was analyzed. The 19 cases involved fracture-dislocation and spinal cord injury at areas of the spine where DISH, ankylosis, spondylosis and/or osteophytes ossified the intervertebral soft tissues causing stiff and brittle joints that were vulnerable to fracture-dislocation by straightening of the spine. Published sled tests at 40 km/h (25 mph) with the 50th Hybrid III showed that peak chest acceleration was 13.5 ± 2.4 g (n=7) and head acceleration was 26.0 ± 12.0 g (n=8). Sled testing at 16 km/h (10 mph) with the BioRID IIg involved T1 x-accelerations of 12.6 ± 2.4 g (n=12) and head x-accelerations of 10.1 ± 0.2 g (n=12). These levels of acceleration are sufficient to fracture the calcified spine of the older occupants without ramping or moving off the support from the seatback and head restraint. A new injury mechanism for spinal fracture-dislocation is described in older occupants with spinal disorders. The occupant remains supported by the relatively upright seatback and high and forward head restraint. The accelerations that bring the occupant up to the delta V are sufficient to fracture-dislocate the calcified spine that tries to straighten in the crash.
机译:在过去的二十年中,座位变得更加强大,并且在后面的冲击中保持更直立。虽然头部限制较高,而且前进的头部和颈部提供支持,但在脊柱疾病的居住者中,胸腔和颈椎的严重致命伤害,如盘(弥漫性发作性骨骼过度症),强直,脊柱病和/或骨折,使脊柱中的关节弄细化。本案例研究解决了脊髓紊乱对骨折脱位和脊髓损伤的影响,与相对直立的座椅相对直立。研究了19个田间事故,其中胸腔和颈椎的严重致命伤害发生,座椅仍然直立或略微倾斜。乘员是膝盖肩带,有些带有皮带预张紧和挂闩板。居民年龄较大,并且脊柱的预先存在疾病,包括糖化脊髓的脊柱,心脏,脊柱病,脊柱病和/或骨赘。总结了崩溃,分析了损伤机制。 19例涉及脊柱区域的骨折脱位和脊髓损伤,其中盘子,吻不紧,脊柱病和/或骨赘骨质化椎间体软组织引起脆弱和脆性关节,脆弱的关节通过矫正脊柱而易受裂缝脱位。与第50 km / h(25英里/小时)发表的SLID测试,第50次杂交III显示峰值胸部加速度为13.5±2.4g(n = 7),头部加速度为26.0±12.0g(n = 8)。 SLED测试以16公里/小时(10英里/小时),涉及12.6±2.4g(n = 12)的T1 X加速度,头X加速度为10.1±0.2g(n = 12)。这些水平的加速度足以破坏较旧的乘员的钙化脊柱,而不斜坡或从座椅靠背和头部束缚中脱离支撑。脊髓紊乱的较旧居民描述了一种新的脊髓骨折脱位机制。乘员仍然受到相对直立的座椅靠背和高端头枕的支持。将乘员带到三角洲v的加速度足以裂开,使试图在碰撞中伸直的钙化脊柱。

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