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NUCLEON DECAY IN GUT AND NON-GUT SUSY MODELS

机译:肠道腐烂在肠道和非肠道Sery模型中

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I first emphasize the importance of searching for nucleon decay in the context of supersymmetric models. The status of minimal SUSY SU(5) model is reviewed, which can be definitively ruled out by a combination of superKamiokande and LEP-2 experiments. Non-minimal models may provide some suppression in the nucleon decay rates, but there is still a good chance for superKamiokande. I point out that the operators suppressed even by the Planck-scale are too large. We need a suppression mechanism for the operators at the level of 10~7, and the mechanism, I argue, may well be a flavor symmetry. A particular example predicts p — if°e+ to be the dominant mode which does not arise in GUT models.
机译:我首先强调在超对称模型的背景下搜索核子衰减的重要性。综述了最小SUSY SU(5)模型的状态,可以通过Superkamiokande和LEP-2实验的组合明确地排除。非最小模型可能在核子衰减率中提供一些抑制,但仍然有很好的机会对Superkamiokande。我指出,甚至通过普拉克级甚至被普拉斯级抑制了太大了。我们需要一个抑制机制,为运营商在10〜7的水平,而我争论的机制可能很可能是一种风味对称性。一个特定的示例预测p - if°E +是肠道模型中不出现的主要模式。

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