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Radiation induced attenuation kinetics in pure-silica-core optical fibers during successive irradiations

机译:辐射诱导连续照射期间纯二氧化硅芯光纤中的衰减动力学

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We present an experimental investigation regarding the 10 keV X-ray radiation induced attenuation (RIA) in the range 2-5 eV (620 - 248 nm) measured on radiation hardened optical fibers. We performed in situ RIA measurements during three irradiation runs having the same dose rate and the same duration. Our data evidence that the radiation induced STH1 and the Cl related defects are mainly responsible for these excess losses in the range 2-3.5 eV during irradiation. This RIA disappears one hour after the irradiation stop. Furthermore, the related RIA kinetic observed during the first irradiation run differs from those observed during the successive runs, these ones being faster during the irradiation and unchanged after it. By contrast, no more differences in the RIA increase were observed in the irradiations successive to the first run. These results indicate that after the first irradiation the initial population and typology of defects, involved in the generation processes, is not restored (irreversible channels). By contrast, the population and typology of the defects present in the fiber after the first transmission recovery is activated by new irradiation and reformed at end of each ones giving rise to reversible channels.
机译:我们介绍了在辐射硬化光纤上测量的2-5 eV(620-248nm)范围内的10keV X射线辐射诱导衰减(RIA)的实验研究。在三次辐照过程中,我们在具有相同剂量率和相同持续时间的三次辐照过程中进行的原位RIA测量。我们的数据证据表明辐射诱导的STH1和CL相关缺陷主要负责在照射期间2-3.5 EV范围内的这些过量损失。在照射停止后,这个RIA消失了一小时。此外,在第一照射期间观察到的相关的RIA动力学与在连续运行期间观察到的那些不同的RIA动力学不同,这些在照射期间更快,并且在它之后不变。相比之下,在第一次运行中,在照射中没有观察到RIA增加的更多差异。这些结果表明,在第一次照射后,初始群体和类型的缺陷类型,涉及生成过程,未恢复(不可逆渠道)。相比之下,通过新的辐射在第一传输回收后激活纤维中存在的缺陷的缺陷和类型学,并在每个人的末端改革,从而产生可逆通道。

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