A device for the introduction of boards 2 in the body of the press, vertical or inclined, of a bale press for goods with short fibers or filaments.

• 1. 用于控制沿圆周包卷在打包机的捆包形成室中的圆柱形捆包的方法和系统以及用于生产沿圆周包卷的捆包的打包机和方法 [P] . 中国专利： CN104244702B . 2016.09.14
• 2. 用于控制沿圆周包卷在打包机的捆包形成室中的圆柱形捆包的方法和系统以及用于生产沿圆周包卷的捆包的打包机和方法 [P] . 中国专利： CN104244702A . 2014-12-24
• 3. A device for introducing clapboard dividing the pressure Channel vertical or inclined Press Baler For Short Fiber products. [P] . ES74131A1 . 1920-09-16

  机译： 一种将隔板分隔成压力的装置，用于短纤维产品的通道垂直或倾斜压力打包机。

• 4. A baler with a device for introduction of part of the boards, in the compression channel [P] . 德国专利： DE334206C . 1921-03-09

  机译： 压捆机，带有用于在压缩通道中引入部分木板的装置

• 5. A baler with a pressed articles with a covering device for the automatically acting on the pressing body upper side openings of the vertical needle feedthrough [P] . 德国专利： DE9315162U1 . 1994-02-03

  机译： 带有被压物品的打捆机，其具有覆盖装置，该覆盖装置用于自动作用于垂直针穿刺装置的压紧主体上侧开口

• 1. Prokaryotic Expression, Purification, Antibody Production of a NH2-Terminal Fragment of mCLCA3 Protein and Analysis of mClCA3 Protein Expression in Asthmatic Mouse Lung [J] . HOU Xia, WU Qing-tian, ZHANG Yu-ping, 高等学校化学研究（英文版） . 2007,第006期
• 2. Antigen Gene Cloning and Expression of HIV-1 for AIDS Vaccine Design Ⅲ. HIV-1 Antigen Gene Cloning, in Vitro Expression and Antibody Induction [J] . ZENG Qingping(曾庆平), FENG Liling(冯丽玲), YANG Ruiyi(杨瑞仪), 中华性传播感染杂志（英文版） . 2002,第003期
• 3. MEASUREMENT OF BALE MOISTURE CONTENT BASED ON BALE COMPRESSION PRESSURE [C] . R.K. Byler, W.S. Anthony Beltwide Cotton Conference . 2004
• 4. Modeling and validation of the baling process in the compression chamber of a large square baler. [D] . Afzalinia, Sadegh. 2005
• 5. Short-Term Depression of Axonal Spikes at the Mouse Hippocampal Mossy Fibers and Sodium Channel-Dependent Modulation [O] . Shunsuke Ohura, Haruyuki Kamiya 2018
• 6. Amitava Krishna Dutt, Oxford India Short Introductions Pathways to Economic Development, Oxford University Press, 2014, 211 pp., Rs 295.Partha Pratim Pal, Oxford India Short Introductions International Trade and India, Oxford University Press, 2014, 244 pp., Rs 295. [O] . Ghosh Sudeshna 2015

• 1. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004） （P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期
• 2. Short-term Femara (letrozole) treatment and suppression of Ki67 expression in postmenopausal endometrial carcinoma [J] . Lloyd H. Smith ,Gary Leiserowitz ,Guibo Xing . 妇产科期刊（英文） . 2013,第3期
• 3. Introduction of a new atmospheric pressure plasma device and application on tomato seeds [J] . Zhuwen Zhou ,Yanfen Huang ,Size Yang . 农业科学（英文） . 2011,第1期
• 4. 一种用于轴向输出相对论磁控管中具有TE11辐射模式的紧凑型输出结构 [C] . Shi Difu ,史迪夫 ,Qian Baoliang . 第十届全国高功率微波学术研讨会 . 2015