Electric motor of low power to mechanisms for winding the clock signals, for the mechanisms of alarm clocks and mechanisms analogous

• 1. 发动机制动方法及用于调节发动机制动功率的装置 [P] . 中国专利： CN105822435B . 2021.05.28
• 2. 发动机制动方法及用于调节发动机制动功率的装置 [P] . 中国专利： CN105822435A . 2016-08-03
• 3. Small electric motor for clock-winding mechanisms, clock striking mechanisms, driving gears and the like [P] . 英国专利： GB380771A . 1932-09-22

  机译： 小型电动马达，用于发条装置，发条装置，驱动齿轮等

• 4. Small electric motor for clock-winding mechanisms, clock striking mechanisms, driving gears and the like, and method of assembling the armature of same [P] . 英国专利： GB380629A . 1932-09-22

  机译： 钟表上条机构，钟表敲击机构，驱动齿轮等的小型电动机及其电枢的组装方法

• 5. Winding device which is continuous and automatic striking mechanism for pendulums and clock signals, particularly applicable to the electric motor to balance the clocks [P] . 法国专利： FR631574A . 1927-12-22

  机译： 连续自动上弦机构，用于摆锤和钟表信号的发条装置，特别适用于平衡钟表的电动机

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• 3. Mechanism of sex hormone level in biological clock disorder induced acne and analysis of TCM Pathogenesis [J] . Zhang Miaoliang, Yang Bilian, Wu Di, 世界中西医结合杂志（英文） . 2016,第002期
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• 5. Cholinergic regulation of circadian rhythms in the SCN: Characterization of signaling mechanisms and the role of intracellular calcium in the directionality of clock resetting. [D] . Artinian, Liana. 2001
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• 1. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004） （P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期
• 2. PATH CONTROL OF A 3-DOF ROBOT MECHANISM ACTUATED WITH LOW-PRESSURE AIR POWERED MOTORS [J] . 张毅 ,邹慧君 ,周双林 . 上海交通大学学报：英文版 . 1999,第1期
• 3. 用于同步高功率固体激光的高精度时钟发生技术研究 [C] . 党钊 ,陈德怀 ,王超 . 第三届全国虚拟仪器学术交流大会 . 2009
• 4. miR--494靶向作用于RIPK1调控NF--κB信号通路在癫痫海马神经元损伤中的作用及机制 [A] . 齐印宝 . 2021