Method of separating cineols from hydrocarbons of similar boiling range

• 1. 一种用于烃类气体的捕集剂及近沸点气体的分离方法 [P] . 中国专利： CN113680172A . 2021-11-23
• 2. 分离旋光羟基桉树脑衍生物的方法 [P] . 中国专利： CN110662749A . 2020-01-07
• 3. A method of Separating 1,8-cineol from 1,8-cineol and - 1.4 - cineole or terpenic hydrocarbons [P] . ES8506202A1 . 1985-11-01

  机译： 从1,8-cineol和-1.4-cineole或萜类烃中分离1,8-cineol的方法

• 4. Methodand apparatus for separating wax/water from hydrocarbon mixture boiling in the lubricating oil range [P] . 美国专利： US4581112A . 1986-04-08

  机译： 从沸程在润滑油范围内的烃混合物中分离蜡/水的方法和设备

• 5. Method and apparatus for separating wax/water from hydrocarbon oil mixture boiling in the lubricating oil range [P] . 美国专利： US4622118A . 1986-11-11

  机译： 从沸程在润滑油范围内的烃油混合物中分离蜡/水的方法和设备

• 1. Correction Method of Light Hydrocarbons Losing and Heavy Hydrocarbon Handling for Residual Hydrocarbon (S1) from Shale [J] . WANG Min, TIAN Shansi, CHEN Guohui, 地质学报（英文版） . 2014,第006期
• 2. Fischer-Tropsch reaction within zeolite crystals for selective formation of gasoline-ranged hydrocarbons [J] . Chengtao Wang, Wei Fang, Liang Wang, 能源化学：英文版 . 2021,第003期
• 3. Fischer-Tropsch reaction within zeolite crystals for selective formation of gasoline-ranged hydrocarbons [J] . Chengtao Wang, Wei Fang, Liang Wang, 天然气化学（英文版） . 2021,第003期
• 4. Boiling Point Distribution of Hydrocarbon Types in Diesel Using Solid-Phase Extraction Followed by GC/FID-EIMS [J] . Li Chengwei, Tian Songbai, Liu Zelong, 中国炼油与石油化工（英文版） . 2008,第003期
• 5. Hydrocarbon Type Analysis of Individual Fractions Depending on the Boiling Range of Lube Base Oil by GCMSD [C] . Myounghan No, Eunkyoung Kim, Hongseok Jung ASMS Conference on Mass Spectrometry and Allied Topics . 2008
• 6. Range-separated hybrids combining a range-separated Perdew-Burke-Ernzerhof functional with exact exchange and random phase approximation correlation. [D] . Irelan, Robert. 2014
• 7. Establishment of normal reference ranges for total and separate glomerular filtration rates in living kidney donors by Gates’ method after renal depth measured using single-photon emission computed tomography/computed tomography [O] . Yushan Wei, Yan Liu, Qiang Li, 2020
• 8. Estimation of boiling and melting points of light, heavy and complex hydrocarbons by means of a modified group vector space method [O] . Li, Hengde, Higashi, Hidenori, Tamura, Kazuhiro 2006

• 1. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004） （P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期
• 2. 低沸点烃类液体空气泡沫灭火试验研究 [C] . 张清林 ,秘义行 ,智会强 . 2011中国消防协会科学技术年会 . 2011
• 3. 近沸点及特殊宽沸点二元物系的热泵精馏分离工艺与控制方案 [A] . 刘兴振 . 2017