A method for carrying out the returns accepted of the diffusion - and press this wae in sugar factories

• 1. 用于解密已加密数据并在存储器空间中定位用于执行的解密数据的方法和设备 [P] . 中国专利： CN100359425C . 2008.01.02
• 2. 用于解密已加密数据并在存储器空间中定位用于执行的解密数据的方法和设备 [P] . 中国专利： CN1656434A . 2005-08-17
• 3. A method and device for returns accepted of shreds the expiration of water from the diffusion in sugar factories [P] . 德国专利： DE712019C . 1941-10-10

  机译： 切碎糖厂因扩散而产生的水分到期的退货方法和装置

• 4. A method for preparing the diffusion wae ser for the returns accepted in the diffusion battery [P] . 德国专利： DE555527C . 1932-07-25

  机译： 为扩散电池中接受的返回值准备扩散水的方法

• 5. Method for heating water in factories diffusion beet sugar and installation for carrying out this method [P] . ES533546A0 . 1985-02-16

  机译： 工厂扩散甜菜糖中加热水的方法和实施该方法的设备

• 1. Gene and protein expression profiles of olfactory ensheathing cells from olfactory bulb versus olfactory mucosa [J] . Yuan-Xiang Lan, Ping Yang, Zhong Zeng, 中国神经再生研究（英文版） . 2022,第002期
• 2. Pseudo-beam method for compressive buckling characteristics analysis of space inflatable load-carrying structures [J] . Changguo Wang, Huifeng Tan, Xingwen Du 力学学报：英文版 . 2009,第005期
• 3. Pseudo-beam method for compressive buckling characteristics analysis of space inflatable load-carrying structures [J] . Changguo Wang, Huifeng Tan, Xingwen Du 力学学报：英文版 . 2009,第005期
• 4. Nogo-A expression in injured spinal cord following human olfactory mucosa-derived olfactory ensheathing cells transplantation [J] . Bin Wang, Qiang Li, Xijing He, 中国神经再生研究（英文版） . 2011,第016期
• 5. Trial development on the "new methods on the simplified numerical expression, using peaks I_p in scalar conductive currents of the first lightning return strokes to the earth I， in peaks E_p and B_p in the extremely far electric fields close to the ground E and extremely far magnetic fields close to the ground B caused by the first lightning return strokes" of downward flashes [C] . Hiroshi Kurita 放電研究会;開閉保護研究会;高電圧研究会 . 2017
• 6. Stochastic reaction-diffusion methods for modeling gene expression and spatially distributed chemical kinetics. [D] . Isaacson, Samuel A. 2005
• 7. The Mini-International Neuropsychiatric Interview is useful and well accepted as part of the clinical assessment for depression and anxiety in primary care: a mixed-methods study [O] . Agneta Pettersson, Sonja Modin, Rolf Wahlström, 1985
• 8. ROLE OF CO-OPERATIVE SUGAR FACTORIES IN RURAL DEVELOPMENT : A CASE STUDY OF DAMAJI SUGAR FACTORY, MANGALWEDA [O] . TODKARI GU 2012
• 9. Al-Ga Interdiffusion, Carbon Acceptor Diffusion, and Hole Reduction in Carbon-Doped Al(0.4)Ga(0.6)As/GaAs Superlattices: The As(4) Pressure Effect [R] . You, H. M., Tan, T. Y., Goesele, U. M., 1993

• 1. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004） （P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期
• 2. 变通或放弃执行现行法律规范是保护弱势群体不得已的一种方法 [C] . 朱应平 . 2010行政法年会 . 2010
• 3. “已实现”方法及多分行方法在证券市场收益率预测中的应用 [A] . 张同辉 . 2016