device for protection of soft tissue injuries in the treatment of oral cavity teeth cutting tools

• 1. 用于正畸治疗中的口腔黏膜保护贴片 [P] . 中国专利： CN211434233U . 2020-09-08
• 2. 一种儿童口腔科治疗用头部固定保护装置 [P] . 中国专利： CN212369250U . 2021-01-19
• 3. DENTAL TREATMENT MOUTH OPENING TOOL CAPABLE OF TREATING TEETH WHILE PROTECTING SOFT TISSUE IN ORAL CAVITY [P] . 韩国专利： KR20160109283A . 2016-09-21

  机译： 在口腔中保护软组织的同时可以治疗牙齿的牙科治疗口开放工具

• 4. DENTAL INSTRUMENT FOR CUTTING SOFT TISSUE CAPABLE OF CUTTING SOFT TISSUE WITHIN THE ORAL CAVITY [P] . 韩国专利： KR20100020273A . 2010-02-22

  机译： 用于在口腔内切开软组织的牙科器械

• 5. DEVICE FOR ISOLATION teeth from oral soft tissue and oral LIQUID [P] . 俄罗斯专利： RU82112U1 . 2009-04-20

  机译： 用于从口腔软组织和口腔液体中分离牙齿的装置

• 1. TREATMENT OF 120 CASES OF SOFT-TISSUE INJURY OF THE SHOULDER-BACK REGION WITH FLOATING NEEDLE [J] . WANG Jian-ping, WANG Ke-hong, WANG Xiu-juan 世界针灸杂志（英文版） . 2005,第004期
• 2. TREATMENT OF 120 CASES OF SOFT-TISSUE INJURY OF THE SHOULDER-BACK REGION WITH FLOATING NEEDLE [J] . 王建萍, 王克红, 王秀娟 世界针灸杂志：英文版 . 2005,第004期
• 3. TREATMENT OF SOFT TISSUE INJURY BY PUNCTURING CLEFT-POINTS [J] . Xu Yunxiang, Chen Guizhen 世界针灸杂志（英文版） . 2001,第004期
• 4. Differentiation and Treatment on Injury of the Sinews (Appendix: the Application of Ruan Zu Zhi Sun Shang Gao Ointment for Soft Tissue Injury in Clinics) [C] . The Third international congress on TCM : Collection of papers . 2001
• 5. The comparison of dental arch forms obtained from teeth, alveolar bone, and the overlying soft tissue. [D] . O'Neil, Patrick D. 2013
• 6. Effect of Oral Contraceptives on Soft Tissue Injury Risk Soft Tissue Laxity and Muscle Strength: A Systematic Review of the Literature [O] . Jaclyn A. Konopka, Lauren J. Hsue, Jason L. Dragoo -1
• 7. THE MANAGEMENT OF THE TEETH, BONE, AND SOFT TISSUES IN PATIENTS RECEIVING TREATMENT FOR ORAL CANCER [O] . JUSTIN J. STEIN, ARTHUR G. JAMES, E. RICHARD KING 1970

• 1. Propagation of Acoustic Waves Caused by the Accelerations of Vibrating Hand-Held Tools in Viscoelastic Soft Tissues of Human Hands and a Mechanobiological Picture for the Related Injuries [J] . Eugen Mamontov ,Viktor Berbyuk . 应用数学与应用物理（英文） . 2017,第10期
• 2. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004） （P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期
• 3. 近、远端双重保护装置在颈动脉狭窄支架治疗中的应用 [C] . 杜志华 ,李宝民 . 2012北京地区神经内科学术年会 . 2012
• 4. PercuSurge远端球囊保护装置在急性心肌梗死介入治疗中作用机制研究 [A] . 郭瑄 . 2006