A method for by means of which comes next of a finely divided solid fuel from a space habitual pressure in a chamber which is under increased pressure

• 1. 一种半导体工艺中腔室压力的控制方法 [P] . 中国专利： CN112695297A . 2021-04-23
• 2. 一种以可变压力关闭腔室中电源电路的方法 [P] . 中国专利： CN108987245A . 2018-12-11
• 3. A method for by means of which comes next of a finely divided solid fuel from a space habitual pressure in a chamber which is under increased pressure [P] . 德国专利： DE1189226B . 1965-03-18

  机译： 接下来是一种方法，通过该方法从压力升高的腔室中的空间惯性压力中分离出细碎的固体燃料

• 4. A method for by means of guidance of a finely divided, solid substance from a space habitual pressure in a chamber which is under increased pressure [P] . 德国专利： DE1496371A1 . 1969-07-03

  机译： 一种用于从处于升高的压力的腔室中的空间惯性压力引导细碎的固体物质的方法

• 5. Device for the continuous by means of guidance of a finely divided, solid substance from a space habitual pressure in a chamber which is under increased pressure [P] . 德国专利： DE1442742A1 . 1969-03-06

  机译： 通过在压力增加的腔室中从空间惯性压力引导细碎的固体物质连续进行的装置

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• 1. Elimination of Fuel Pressure Fluctuation and Multi-injection Fuel Mass Deviation of High Pressure Common-rail Fuel Injection System [J] . LI Pimao ,ZHANG Youtong ,LI Tieshuan . 中国机械工程学报 . 2015,第002期
• 2. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004） （P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期
• 3. 反应堆压力容器下腔室交混特性的数值模拟方法研究 [C] . SUI Xi ,眭曦 ,朱勇辉 . 中国原子能科学研究院第30届“五四”青年学术报告会 . 2018
• 4. 离心泵泵腔和平衡腔液体压力的试验及计算方法的研究 [A] . 沈小波 . 2016