method and arrangement to increase the konturenschaerfe a display durchzweiseitige aperturkorrektur of bildschreibenden fernsehsignals

• 1. 用于分析物体轮廓图像的方法与图像处理装置 ,用于检测物体的方法与图像处理装置 ,工业视觉设备 ,智能相机,图像显示器 ,安全系统 ,以及计算机程序产品 [P] . 中国专利： CN1867928A . 2006-11-22
• 2. 利用同调取样技术增加图像像素清晰度的装置及其方法 [P] . 中国专利： CN100472605C . 2009.03.25
• 3. method and arrangement to increase the konturenschaerfe a display durchzweiseitige aperturkorrektur of bildschreibenden fernsehsignals [P] . 法国专利： FR1586139A . 1970-02-13

  机译： 通过图像写入电视信号的双面孔径校正来增加显示器的轮廓清晰度的方法和装置

• 4. Method and arrangement for the increase of the konturenschaerfe of a television picture by means of two side aperture correction of the image read ends television signal. [P] . 德国专利： DE1537111B2 . 1974-05-16

  机译： 通过对读取的电视信号的两侧孔径校正来增加电视画面的动感的方法和装置。

• 5. Method and arrangement for the increase of the konturenschaerfe of a television picture by means of two side aperture correction of the image read ends television signal. [P] . 德国专利： DE1537111A1 . 1969-08-14

  机译： 通过对读取的电视信号的两侧孔径校正来增加电视画面的动感的方法和装置。

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• 2. Optimization of Adsorption Conditions of Papain on Cross-Linked Phage-Displayed Heptapeptide Matrix Using Response Surface Methodology [J] . YANG Huiqin, CHEN Tianxiang, LIU Lin, 东华大学学报（英文版） . 2017,第001期
• 3. Increase of brightness and transmission efficiency in fiat panel display through serial synchronous scanning mode with double buffers [J] . 陈章进, 陈峰, 冉峰, 上海大学学报：英文版 . 2007,第003期
• 4. Methods of creation of thin-film oxide phosphors with increased conductivity for filed emission displays [C] . V.Bondar, V.Vasyltsiv, M.Vasyliv, International symposium on physics and chemistry of luminescent materials . 1999
• 5. Displaying geographic information: Efficient methods for raster displays. [D] . McCroan, Keith Duane. 1990
• 6. Oligonucleotides and ND-FISH Displaying Different Arrangements of Tandem Repeats and Identification of Dasypyrum villosum Chromosomes in Wheat Backgrounds [O] . Zhiqiang Xiao, Shuyao Tang, Ling Qiu, 2017
• 7. Modified Method of Increasing of Reconstruction Quality of Diffractive Optical Elements Displayed with LC SLM [O] . Krasnov V.V., Cheremkhin P.A., Erkin I. Yu., 2015

• 1. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004） （P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期
• 2. TNF-a induce the F-actin arrangement and permeability increase in endothelial cells by RhoA-ERK1/2 pathway [J] . YAN Cheng-huiHAN Ya-lingTAO JieDENG JieLUAN BoWU Guang-zheZHANG Xiao-lin (Department of CardiologyCardiovascular Institute of PLA Shenyang Northern HospitalShenyang 310016China) . 岭南心血管病杂志 . 2011,第S1期
• 3. 提高印花轮廓清晰度的途径与方法 [C] . 胡木升 . 2016天创杯环保节能与印染新技术交流会 . 2016
• 4. 平板显示器运动图像边缘清晰度的研究 [A] . 伞光宇 . 2010