A method for producing a a 93 - to 100 strength alcoholysis, which is subject to polyvinyl alcohol with a low content of in cold water soluble proportions

• 1. 聚乙烯醇生产中醇解废液的分离回收方法及设备 [P] . 中国专利： CN103373919A . 2013-10-30
• 2. 聚乙烯醇生产中醇解废液的分离回收装置 [P] . 中国专利： CN203333539U . 2013.12.11
• 3. PRODUCTION OF 93-100 ALCOHOLYZED POLYVINYL ALCOHOL HAVING LOW COLD WATER SOLUBLES CONTENT AND IMPROVED SLURRYING PROPERTIES [P] . 美国专利： US3654247A . 1972-04-04

  机译： 制备具有低冷水含量和改善灌浆性能的93-100％醇解聚乙烯醇

• 4. PRODUCTION OF 93-100 ALCOHOLYZED POLYVINYL ALCOHOL HAVING LOW COLD WATER SOLUBLES CONTENT [P] . CA873409A . 1971-06-15

  机译： 具有低冷水含量的93-100％醇解聚乙烯醇的生产

• 5. procedure for the manufacture of a 93 to 100 alkoholyse polyvinylalkohols subject with low content of cold water loeslichen shares [P] . 德国专利： DE1909168C3 . 1980-09-11

  机译： 制备低含量Loeslichen含量为93％至100％的醇溶性聚乙烯醇的方法

• 1. A METHOD FOR CREATING INTERNAL GEOMETRIES IN INJECTION MOLDED PARTS USING WATER SOLUBLE POLYVINYL ALCOHOL (PVOH) INSERTS [C] . Jason D. McNulty, Hrishikesh Kharbas, Cyrus Thompsonac Technical Conference of the Society of Plastics Engineers . 2015
• 2. Evaluation of extraction method and characterization of water soluble organics from produced water by Fourier transform ion cyclotron resonance mass spectrometry. [D] . Lewis, Adam. 2013
• 3. PMMA-Etching-Free Transfer of Wafer-scale Chemical Vapor Deposition Two-dimensional Atomic Crystal by a Water Soluble Polyvinyl Alcohol Polymer Method [O] . Huynh Van Ngoc, Yongteng Qian, Suk Kil Han, -1
• 4. Studies on the Swelling of Polyvinyl Alcohol. (VI) : Swelling of Films Prepared from Water-Soluble and Insolu b l e Parts of Polyvinyl Alcohol Films [O] . Sone Yasuo, Sakurada Ichiro 1958
• 5. Method of cross-linking polyvinyl alcohol and other water soluble resins [R] . Phillipp, W. H., May, C. E., Hsu, L. C. 1980

• 1. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004） （P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期
• 2. 生物质醇解生产高值化学品:醇解轻油蒸馏过程研究 [C] . 邹献武 ,秦特夫 . 第四届全国生物质材料科学与技术学术研讨会 . 2011
• 3. 一种层状双氢氧化物的制备及其与聚乙烯醇复合薄膜的阻燃性能研究 [A] . 马佳骏 . 2018