procedure for the manufacture of a 93 to 100 alkoholyse polyvinylalkohols subject with low content of cold water loeslichen shares

• 1. 一种测定聚乙烯醇中总氮含量的样品处理方法以及聚乙烯醇中总氮含量的测定方法 [P] . 中国专利： CN110954647A . 2020-04-03
• 2. 一种低甲醇含量的聚乙烯醇水溶液及其制备方法 [P] . 中国专利： CN102786698B . 2014.05.14
• 3. procedure for the manufacture of a 93 to 100 alkoholyse polyvinylalkohols subject with low content of cold water loeslichen shares [P] . 德国专利： DE1909168B2 . 1980-01-17

  机译： 制备低含量Loeslichen含量为93％至100％的醇溶性聚乙烯醇的方法

• 4. procedure for the manufacture of a 93 to 100 alkoholyse polyvinylalkohols subject with low content of cold water loeslichen shares [P] . 德国专利： DE000001909168A . 1969-09-11

  机译： 含量低的冷水Loeslichen含量为93至100％的醇溶性聚乙烯醇的生产程序

• 5. A method for producing a a 93 - to 100 strength alcoholysis, which is subject to polyvinyl alcohol with a low content of in cold water soluble proportions [P] . 德国专利： DE1909168A1 . 1969-09-11

  机译： 一种生产93％至100％强度的醇解的方法，该醇解受制于冷水可溶物比例低的聚乙烯醇

• 1. Influence of the northern Yellow Sea Cold Water Mass on picoplankton distribution around the Zhangzi Island, northern Yellow Sea [J] . ZHAO Li, ZHAO Yanchu, DONG Yi, 海洋学报（英文版） . 2018,第005期
• 2. The temporal and spatial variability of the Yellow Sea Cold Water Mass in the southeastern Yellow Sea, 2009-2011 [J] . OH Kyung-Hee, LEE Seok, SONG Kyu-Min, 海洋学报（英文版） . 2013,第009期
• 3. Diel vertical migration of the copepod Calanus sinicus before and during formation of the Yellow Sea Cold Bottom Water in the Yellow Sea [J] . KANG Jung-Hoon, SEO MinHo, KWON Oh Youn, 海洋学报（英文版） . 2013,第009期
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• 6. Similar Genetic Architecture with Shared and Unique Quantitative Trait Loci for Bacterial Cold Water Disease Resistance in Two Rainbow Trout Breeding Populations [O] . Roger L. Vallejo, Sixin Liu, Guangtu Gao, 2017
• 7. Proposal for a European Parliament and Council Directive amending Directive 92/50/EEC relating to the coordination of procedures for the award of public service contracts Directive 93/36/EEC coordinating procedures for the award of public supply contracts, and Directive 93/37/EEC concerning the coordination of procedures for the award of public work contracts. Proposal for a European Parliament and Council Directive amending Directive 93/38/EEC coordinating the procurement of entities operating in the water, energy, transport and telecommunications sectors. COM (95) 107 final, 29 March 1995 [O] . 1995
• 8. Cost Reduction in Irrigation Well Pump Application. Part II: User's Manual for Detailed Design of Radial- or Mixed-Flow Centrifugal Rotors for Deep-Well Pumps Using Cold Water (Less than 100 Degrees F) and with Sufficient Inlet Head to Avoid Cavitation [R] . Stanitz, J. D. 1981

• 1. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004） （P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期
• 2. The diel vertical migration of the sound-scattering layer in the Yellow Sea Bottom Cold Water of the southeastern Yellow Sea: focus on its relationship with a temperature structure [J] . LEE Hyungbeen ,CHO Sungho ,KIM Woongseo . 海洋学报：英文版 . 2013,第9期
• 3. 白桦树皮桦木醇含量测定的样品制备方法优化 [C] . 王倩 ,王洋 ,阎秀峰 . 第八届中国林业青年学术年会 . 2008
• 4. 低聚合度低醇解度聚乙烯醇的制备研究 [A] . 许东颖 . 2003