method for storing in a memory a farbfernsehsignals lower upper limiting frequency than that of the farbfernsehsignals

• 1. 一种液相色谱法分离测定右美沙芬与左美沙芬的方法 [P] . 中国专利： CN111323529A . 2020-06-23
• 2. MRS成像法中调节激励信号频带中心频率和接收带宽而抑制水信号且增强代谢物信号方法 [P] . 中国专利： CN108700638B . 2021.07.16
• 3. method for storing in a memory a farbfernsehsignals lower upper limiting frequency than that of the farbfernsehsignals [P] . 德国专利： DE000001537108A . 1969-12-18

  机译： 在存储器中存储法呢芬信号的上限频率比法呢芬信号的上限频率低的方法

• 4. method for storing in a memory farbfernsehsignalen with lower frequency than that of the upper farbfernsehsignals [P] . 德国专利： DE000001462599A . 1969-02-20

  机译： 在存储器中存储频率低于上法铁信号的频率的方法

• 5. method for storing tubes signals to memory with lower upper limiting frequency than that of the notice signal [P] . AT287092B . 1971-01-11

  机译： 将电子管信号存储到存储器的方法，其上限频率低于通知信号的上限频率

• 1. THE APPLICATION OF WEINSTEIN-CHIEN′S METHOD——THE UPPER AND LOWER LIMITS OF FUNDAMENTAL FREQUENCY OF RECTANGULAR PLATES WITH EDGES ARE THE MIXTURE OF SIMPLY SUPPORTED PORTIONS AND CLAMPED PORTIONS [J] . 陈政清 应用数学和力学：英文版 . 1984,第003期
• 2. A Lower and Upper Bound Method for Complex System Reliability Evaluation [J] . WEI Zhan-ming, ZHOU Fan, CHEN Yao-wu 东华大学学报（英文版） . 2011,第005期
• 3. A Mechanism Responsible For The Observation Of Symmetric Lower Hybrid Sidebands And A Lower Frequency Mode In The Upper Ionosphere [C] . Dalkir, Y.R., Lee, . 1992
• 4. Radio frequency interference mitigation methods applied to the LAAS multipath limiting antenna for system availability improvements. [D] . Sharawi, Mohammad Said. 2006
• 5. Association of lymph-node antigens with lower Gag-specific central-memory and higher Env-specific effector-memory CD8+ T-cell frequencies in a macaque AIDS model [O] . Hiroshi Ishii, Saori Matsuoka, Takushi Nomura, -1
• 6. A Study on Multiple Storied Forests : 1. Stand structure of an upper storey and growth of lower-storey trees (Forestry Experiment Station) [O] . 平田 永二, 山盛 直, 安里 練雄, 1995

• 1. Discrimination between upper versus lower airway components to the rise of total airway resistance measured by Pennock’s method after nasal irritant challenge [J] . Zuzana Biringerova ,Silvia Gavliakova ,Eva Hanuskova . 分子和综合生理学期刊（英文） . 2013,第3期
• 2. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004） （P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期
• 3. Biorational control methods for protection of stored grain legumes against bruchid beetles [J] . Takashi Yamane . 农业科学（英文） . 2013,第12期
• 4. 用差频法测量频率信号 [C] . 刘晓明 . 第五届全国电路理论及应用学术研讨会 . 1997
• 5. 极限分析上限法在加筋土结构物稳定性分析中的应用 [A] . 李秀娟 . 2008