method for storing tubes signals to memory with lower upper limiting frequency than that of the notice signal

• 1. 一种以低于奈奎斯特频率的采样频率进行信号采集方法 [P] . 中国专利： CN103346798B . 2016.07.06
• 2. 一种以低于奈奎斯特频率的采样频率进行信号采集方法 [P] . 中国专利： CN103346798A . 2013-10-09
• 3. method for storing in a memory a farbfernsehsignals lower upper limiting frequency than that of the farbfernsehsignals [P] . 法国专利： FR1596402A . 1970-06-15

  机译： 在存储器中存储法呢芬信号的上限频率比法呢芬信号的上限频率低的方法

• 4. method for storing in a memory a farbfernsehsignals lower upper limiting frequency than that of the farbfernsehsignals [P] . 德国专利： DE000001537108A . 1969-12-18

  机译： 在存储器中存储法呢芬信号的上限频率比法呢芬信号的上限频率低的方法

• 5. A method for storing a color television signal in a memory having a lower upper limit frequency than that of the color television signal [P] . 德国专利： DE1537108A1 . 1969-12-18

  机译： 将彩色电视信号存储在具有比彩色电视信号的上限频率低的上限频率的存储器中的方法

• 1. Sensibility to Changes of Vibrational Modes of Excited Electron: Sum Frequency Signals Versus Difference Frequency Signals* [J] . GU An-Na, LIANG Xian-Ting 理论物理通讯（英文版） . 2011,第003期
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• 3. An Improved Lower Order Method of Modal Parameter Estimation for Offshore Structures Using Reconstructed Signals [J] . LIU Fushun, QIN Junfei, LI Huajun, 中国海洋大学学报（英文版） . 2015,第006期
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• 5. New time-frequency domain pitch estimation methods for speech signals under low levels of SNR. [D] . Shahnaz, Celia. 2009
• 6. Multiple Drug Treatments That Increase cAMP Signaling Restore Long-Term Memory and Aberrant Signaling in Fragile X Syndrome Models [O] . Catherine H. Choi, Brian P. Schoenfeld, Aaron J. Bell, 2016
• 7. Multiple Drug Treatments That Increase cAMP Signaling Restore Long-Term Memory and Aberrant Signaling in Fragile X Syndrome Models [O] . Catherine H. Choi, Brian P. Schoenfeld, Aaron J. Bell, 2016
• 8. Amplification of Electrical Signals with Molecule-Based Transistors: Power Amplification Up to a Kilohertz Frequency and Factors Limiting Higher Frequency Operation [R] . Lofton, E. P., Thackeray, J. W., Wrighton, M. S. 1986

• 1. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004） （P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期
• 2. Early detection of sudden cardiac death by using classical linear techniques and time-frequency methods on electrocardiogram signals [J] . Elias Ebrahimzadeh ,Mohammad Pooyan . 生物医学工程（英文） . 2011,第11期
• 3. 一种改善DDS频率上限与杂散电平的方法 [C] . 谭姝静 ,费元春 . 第二届DDS技术与应用研讨会 . 2001
• 4. 谷丙转氨酶低于2倍正常上限的慢乙肝患者肝组织病理学改变及相关因素分析 [A] . 杨瑞 . 2014