METHOD OF MEASURING THE VOLUME RESISTIVITY OF THIN,SOLID DIELECTRIC MATERIAL UTILIZING THE DECAY RATES OF A NUMBER OF MEASURED DECAY INTERVALS

• 1. 利用建筑结构振动衰减率评估建筑物二次辐射噪声的方法 [P] . 中国专利： CN107167230B . 2019.06.28
• 2. 利用运行列车为激励的钢轨振动衰减率的测试方法 [P] . 中国专利： CN107192763B . 2019.06.28
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  机译： 利用辐射衰减曲线和确定热中子衰减时间常数的方法

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  机译： 长衰变发光材料及其制造方法

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  机译： 长衰变发光材料及其制造方法

• 1. DECAY RATE OF FOURIER TRANSFORMS OF SOME SELF-SIMILAR MEASURES [J] . Xiang GAO, Jihua MA 数学物理学报（英文版） . 2017,第006期
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• 4. ANAPHASE CHROMOSOME VELOCITY IS NOT PROPORTIONAL TO THE CONCENTRATION OF ORIENTED MATERIAL, INCLUDING MICROTUBULES, IN CHROMOSOMAL SPINDLE FIBRES OR TO THE RATE OF DECAY OF THE ORIENTED MATERIAL. [D] . SCHAAP, CATHERINE JO. 1983
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• 1. Effects of a Periodic Decay Rate on the Statistics of Radioactive Decay: New Methods to Search for Violations of the Law of Radioactive Change [J] . M. P. Silverman . 现代物理（英文） . 2015,第11期
• 2. Self-heating Probe Instrument and Method for Measuring High Temperature Melting Volume Change Rate of Material [J] . WANG Junwei ,WANG Zhiping ,LU Yang . 中国机械工程学报 . 2013,第002期
• 3. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004） （P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期
• 4. 设备衰减率对太阳能光伏发电利用小时的影响分析 [C] . . 2017第十九届中国科协年会 . 2017
• 5. 计算QBD衰减率的数值方法 [A] . 徐喻军 . 2010