A differential gear mechanism controlling the torque or desequilibrador and improvement in the process for dividing torque.

• 1. 具有改进的最大扭矩和扭矩密度的差速器 [P] . 中国专利： CN101968112B . 2015.07.22
• 2. 具有改进的最大扭矩和扭矩密度的差速器 [P] . 中国专利： CN101968112A . 2011-02-09
• 3. TORQUE DIVIDING MECHANISM FOR DIFFERENTIAL GEAR [P] . 日本专利： JPH06297977A . 1994-10-25

  机译： 差速器的扭矩分配机构

• 4. Transmission, vehicle, hybrid vehicle and unit for controlling and for switching between first and second power transmission processes consists of electric motor, a differential gearing mechanism and power coupling mechanism [P] . 德国专利： DE10004812A1 . 2000-08-17

  机译： 变速器，车辆，混合动力车辆以及用于控制第一动力传递过程和第二动力传递过程的单元，包括电动机，差速齿轮机构和动力耦合机构。

• 5. Improvements in differential gear control mechanism [P] . 英国专利： GB157888A . 1921-09-01

  机译： 差动齿轮控制机构的改进

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• 7. Processing Efficiency of Divided Spatial Attention Mechanisms in Human Visual Cortex [O] . Stephanie A. McMains, David C. Somers 2005
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• 1. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004） （P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期
• 2. 一种平衡机构输入扭矩的主动控制装置 [C] . 高航 ,姚燕安 ,查建中 . 2005年中国机械工程学会年会 . 2005
• 3. 大扭矩复合齿轮马达瞬态扭矩及实验研究 [A] . 宋艳亮 . 2006