device and method for the establishment of immunity to noise for control signals in elektrostatografische establishments.

• 1. 信息记录介质、信息记录装置和方法、息重放装置和方法、信信息录放装置和方法、用于控制记录或重放的计算机程序,以及包括控制信号的数据结构 [P] . 中国专利： CN1739291A . 2006-02-22
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• 3. Method of improving noise immunity in a signal processing apparatus, and a signal processing apparatus having improved noise immunity [P] . 美国专利： US9503055B2 . 2016-11-22

  机译： 改善信号处理设备中的抗干扰性的方法以及具有改善的抗干扰性的信号处理设备

• 4. Method of improving noise immunity in a signal processing apparatus, and a signal processing apparatus having improved noise immunity [P] . 美国专利： US8975953B2 . 2015-03-10

  机译： 改善信号处理设备中的抗干扰性的方法以及具有改善的抗干扰性的信号处理设备

• 5. METHOD OF IMPROVING NOISE IMMUNITY IN A SIGNAL PROCESSING APPARATUS, AND A SIGNAL PROCESSING APPARATUS HAVING IMPROVED NOISE IMMUNITY [P] . 美国专利： US2015318841A1 . 2015-11-05

  机译： 改善信号处理装置中的噪声抗扰性的方法以及具有改善的噪声抗扰性的信号处理设备

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• 1. Estrogen signaling effects on muscle-specific immune responses through controlling the recruitment and function of macrophages and T cells [J] . Liao Zhaohong . 解剖学杂志 . 2021,第S01期
• 2. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004） （P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期
• 3. 决策树方法在静息心电图ST-T改变及静息时心率与冠状动脉疾病的严重程度的关系分析的应用 [C] . . 2016年中国生物统计学术年会 . -1
• 4. 用于轻度认知障碍患者诊断的静息态脑电信号识别方法 [A] . 李鹏 . 2019