METHOD OF SEPARATING THE MERCURY FROM THE SOLUTIONS PARTI. ANALYZED BY THE POLAROGRAPHY

• 1. 食用油中甾醇的电喷雾离子化-四极杆-飞行时间串联质谱分析方法 [P] . 中国专利： CN102967647B . 2015.08.05
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• 3. METHOD FOR ANALYZING PLATING SOLUTION AND PLATING METHOD USING THE SAME CAPABLE OF SEPARATING ONLY A POLYMERIC MATERIAL FROM PLATING SOLUTION USING AN ORGANIC-SOLVENT EXTRACTION TECHNIQUE AND PRECISELY ANALYZING DECREASE IN THE CONCENTRATION OF A POLYMERIC MATERIAL [P] . 韩国专利： KR20120006884A . 2012-01-19

  机译： 解析溶液的方法和利用有机溶剂萃取技术从沉积溶液中分离聚合物的能力相同的沉积方法，并精确地分析聚合物材料浓度的下降

• 4. METHOD OF DEPOSITING AND SEPARATING MERCURY FROM SOLUTION CONTAINING MERCURY [P] . 日本专利： JPS5429293B2 . 1979-09-21

  机译： 从含汞溶液中分离汞的方法

• 5. METHOD OF DEPOSITING AND SEPARATING MERCURY FROM SOLUTION CONTAINING MERCURY [P] . 日本专利： JPS5273121A . 1977-06-18

  机译： 从含汞溶液中分离汞的方法

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• 7. A High-Sensitive Polarography in Highly Viscous Supporting Electrolyte Solution Using Stationary Mercury Electrode [O] . Yasushl MASHIKO, Noboru HOSOYA, Keiichi ITO 1962
• 8. THE DETERMINATION OF MERCURY, COFFER, AND ZINC BY DERIVATIVE POLAROGRAPHY - APPLICATION TO SOLUTIONS OF URANYL SULFATE CONTAINING- CORROSION PRODUCTS [R] . R. G. Ball, D. L. Manning, 0. Menis 1959

• 1. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004） （P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期
• 2. The Challenge and Its Solution When Determining Biogeochemically Reactive Inorganic Mercury(RHg):Getting the Analytical Method Right [J] . Lian Liang ,Milena Horvat ,John Alvarez . 美国分析化学（英文） . 2013,第11期
• 3. 水介质中丰索磷的极谱性能及其在汞滴上的吸附 [C] . 王维德 . 第三届全国电分析化学学术会 . 1987
• 4. 汞砷蒸气发生新方法及其在原子光谱分析中的应用研究 [A] . 杨清华 . 2011