A method for increasing the rigidity of warm shaped articles from being removed from the mold

• 1. 一种将塑料制品从模具中取出装置 [P] . 中国专利： CN109016407A . 2018-12-18
• 2. 一种注塑制品中金属嵌件的取出装置及其取出方法 [P] . 中国专利： CN103419302B . 2015.09.30
• 3. RIGIDITY IMPROVEMENT METHOD FOR PRESS-MOLDED ARTICLE, PRESS-MOLDING DIE, PRESS-MOLDED ARTICLE, AND PRODUCTION METHOD FOR PRESS-MOLDED ARTICLE [P] . 世界知识产权组织专利： WO2019187864A1 . 2019-10-03

  机译： 压模制品的刚性改进方法，压模，压模制品以及压模制品的生产方法

• 4. METHOD FOR REMOVING BURR FROM MOLDED ARTICLE, BURR REMOVING TABLE FOR MOLDED ARTICLE, AND BURR REMOVING APPARATUS FOR MOLDED ARTICLE [P] . 世界知识产权组织专利： WO2015136793A1 . 2015-09-17

  机译： 从模制件去除毛刺的方法，模制件的毛刺去除台和模制件的毛刺去除装置

• 5. DOUBLE MOLDED PLASTIC FUEL TANK INSERTING A STRUCTURE FOR REINFORCING RIGIDITY INTO BETWEEN A FIRST MOLDED ARTICLE AND A SECOND MOLDED ARTICLE, AND A MANUFACTURING METHOD THEREOF [P] . 韩国专利： KR20130033818A . 2013-04-04

  机译： 在第一模制件和第二模制件之间插入用于增强刚性的结构的双模制塑料燃料罐及其制造方法

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• 1. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004） （P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期
• 2. Mold Thermo-Mechanical Rigidity Criterion for Surface Quality of Continuous Casting of Steel [J] . Mostafa Omar El-Bealy . 材料科学与应用期刊（英文） . 2013,第1期
• 3. Study and Application of Mold Filling Simulation of Shaped Castings [J] . Baicheng LIU ,Wei QIU ,Houfa SHEN . 材料科学技术：英文版 . 1997,第2期
• 4. 一种关于乳及乳制品中乳铁蛋白的检测方法 [C] . 徐向峰 ,袁凤琴 ,李照 . 2015中国乳制品工业协会第二十一次年会 . 2015
• 5. 一种在WCDMA系统中增加认知功能的方法与性能分析 [A] . 马瑞 . 2011