Method of detecting a misload of a tube into a volumetric infusion pump

• 1. 输液泵输液状态检测方法、输液泵、医疗设备及存储介质 [P] . 中国专利： CN114126683A . 2022-03-01
• 2. 输液泵及输液泵气泡检测方法 [P] . 中国专利： CN113134129A . 2021-07-20
• 3. Method of detecting a misload of a tube into a volumetric infusion pump by detecting whether interstice is closed and then reversing cam rotation to unload tube [P] . NZ336736A . 2000-03-27

  机译： 通过检测空隙是否闭合然后反转凸轮旋转以卸载管子来检测将管子误装到容积式输液泵中的方法

• 4. Method of detecting a misload of a tube into a volumetric infusion pump [P] . AU725739B2 . 2000-10-19

  机译： 检测进入容积式输液泵的管的不正确负载的方法

• 5. A method of loading a tube into a volumetric infusion pump [P] . AU739019B2 . 2001-10-04

  机译： 一种将管子装入容积输液泵的方法

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• 3. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004） （P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期
• 4. 输液泵性能检测之流速检测方法研究与改进 [C] . 李静 . 中华医学会医学工程学分会第十五次全国学术年会 . 2015
• 5. 公共用电区域恶性负载智能检测方法研究 [A] . 秦昱 . 2021