Dough-handling systems with elevator proofing-rack mechanisms and flip-slat oven-entry mechanisms for manufacturing bakery products and methods therein

• 1. 烘焙产品样低蛋白食品制造用面团、预混料、含有其的烘焙产品样低蛋白食品及其制造方法 [P] . 中国专利： CN111465324A . 2020-07-28
• 2. 电梯承载机构、用于此类承载机构的制造方法以及具有此类电梯承载机构的电梯设备 [P] . 中国专利： CN102264623B . 2013.09.04
• 3. COATING MATERIAL FOR BAKERY PRODUCT, COMPOSITION FOR COATING MATERIAL FOR BAKERY PRODUCT, BAKERY PRODUCT, AND MANUFACTURING METHOD OF BAKERY PRODUCT [P] . 日本专利： JP2019092456A . 2019-06-20

  机译： 面包制品用涂料，面包制品用涂料组合物，面包制品以及面包制品的制造方法

• 4. BAKERY PRODUCT CONDITIONER FOR WATER-ROUX STARTER, WATER-ROUX STARTER FOR BAKERY PRODUCT, BAKERY PRODUCT AND MANUFACTURING METHOD OF WATER-ROUX STARTER FOR BAKERY PRODUCT [P] . 日本专利： JP2016187310A . 2016-11-04

  机译： 糊状发酵剂的烘培产品调理剂，糊状发酵剂的水培发酵剂，烘培产品和糊状发酵剂的水培发酵剂的制造方法

• 5. two or mehrphasiges mechanisms and procedures for preservation of bakery products. [P] . AT139439B . 1934-11-10

  机译： 两种或多种用于烘焙产品保存的方法和程序。

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• 1. A Review on Photovoltaic Systems: Mechanisms and Methods for Irradiation Tracking and Prediction [J] . Hermes José Loschi ,Yuzo Iano ,Julio León . 智能电网与可再生能源（英文） . 2015,第7期
• 2. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004） （P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期
• 3. A Study on Propagation Mechanism of Fracture Systems in Rock Masses by Discontinuity Displacement Method [J] . Tang HuimingChina University of Geosciences . Wihan 430074 . 地球科学学刊：英文版 . 1993,第1期