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Method and Device for Detecting Objects from Depth-Resolved Image Data

机译：从深度分辨图像数据中检测物体的方法和装置

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摘要

The invention relates to a method and a device for detecting objects. The method comprises the following steps:forming at least one object from a depth image of a 3D cameraevaluating and classifying the one or more objects (2, 3) in a 2D image (5) which corresponds to the at least one formed object in the depth image (1),possibly dividing the at least one object (1) formed from the depth image into a plurality of individual objects (2, 3) while taking into account the classification of the one or more objects (2, 3) in the 2D image (5).;The method and the device can be used in particular within the framework of a driver assistance system for detecting objects in a vehicle environment.

机译：本发明涉及一种用于检测物体的方法和装置。该方法包括以下步骤：从3D相机的深度图像中形成至少一个对象评估并分类一个或多个对象（ 2、3 ）在2D图像（ 5 ）中，该图像与深度图像（ 1 ）中至少一个形成的对象相对应，可能将深度图像形成的至少一个对象（ 1 ）分为多个单独的对象（ 2、3 ），同时考虑到2D图像（ 5 ）中一个或多个对象（ 2、3 ）的分类。 ;该方法和设备尤其可以在驾驶员辅助系统的框架内用于检测车辆环境中的物体。 展开▼

著录项

公开/公告号US2016371549A1

专利类型

公开/公告日2016-12-22

原文格式PDF

申请/专利权人 CONTI TEMIC MICROELECTRONIC GMBH;
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申请/专利号US201414902176

发明设计人 ROBERT THIEL;ALEXANDER BACHMANN;
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申请日2014-09-04

分类号G06K9;B60R1;G06K9/34;H04N13/02;G06K9/62;

国家 US

入库时间 2022-08-21 13:48:15

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5. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004）（P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期

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