A method for increasing production economics chickens when irradiated with laser light 'Matrix' lamps and DRT-400 and-500 DNESG

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• 3. A method for increasing the viability of the chick radiant laser energy "Matrix" and lamp DRT-400 [P] . 俄罗斯专利： RU2015154167A . 2017-06-26

  机译： 一种提高小鸡辐射激光能量“矩阵”和灯DRT-400的生存能力的方法

• 4. A method of stimulating blood hematopoietic laser light bird "Matrix" and lamp DRT-400 [P] . 俄罗斯专利： RU2015154178A . 2017-06-21

  机译： 一种刺激血液造血激光鸟“基质”和灯DRT-400的方法

• 5. A method of stimulating the biochemical blood of the bird indicators light la Zera "Matrix" and lamp DRT-400 [P] . 俄罗斯专利： RU2015154165A . 2017-06-21

  机译： 一种刺激鸟类指标la Zera“ Matrix”和DRT-400灯生化血液的方法

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• 1. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004） （P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期
• 2. Expression of matrix metalloproteinases and tissue inhibitor metalloproteinases increases in X-irradiated rat ileum despite the disappearance of CD8a T cells [J] . Carine Strup-Perrot ,Marie-Catherine Vozenin-Brotons ,Marie Vandamme . 世界胃肠病学杂志：英文版 . 2005,第40期
• 3. 测量低压汞灯254nm辐通量的Keitz方法 [C] . 张源 ,孙鸣华 ,朱绍龙 . 第五届全国电光源科技研讨会 . 2009
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