FIELD: medicine.;SUBSTANCE: invention relates to medicine and can be used to diagnose secondary sensorineural hearing loss due to the reduced perilymphatic pressure of the labyrinth. Conduct audiometric control of the acuity of the patient's hearing when sitting in a soundproofed room. Determine initial thresholds for the sound perception of the patient by air conduction throughout the tone scale. Then the patient maximum tightly presses head to the sternum and holds the head in this position for 55–65 seconds. Immediately against this background, the patient determines the presence or absence of a change in his sound perception thresholds by air conduction throughout the tone scale at different frequencies of sound. Further patient additionally in the prone position continuously performs within 60–65 seconds a deep breath-exhalation with a respiratory rate of 20–22 per minute. After stabilization of low intracranial pressure for 60–65 seconds, the patient's presence or absence of change in the sound perception thresholds for air conduction across the entire tone scale at different sound frequencies at a step of 5 dB. If a fact of lowering the thresholds of sound perception by air conduction of the patient by 10 dB or more is found on at least two different frequencies of sounding with increasing liquor pressure, as well as simultaneous detection of an increase in the sound perception thresholds for air conduction of the patient by 10 dB and more at not less than two different frequencies of sounding with a decrease in intracranial pressure, the patient is diagnosed with a secondary sensorineural hearing loss due to a lower perilymphatic pressure of the labyrinth.;EFFECT: method allows to diagnose, with a high degree of reliability, secondary sensorineural hearing loss due to a lower perilymphatic labyrinth pressure by determining the change in its sound perception thresholds by air conduction throughout the tone scale at different frequencies of sound when the patient performs the maximum tightening of the head to the sternum and its retention and the patient's deep breaths-exhalations.;1 cl, 3 ex
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机译：技术领域本发明涉及医学，并且可以用于诊断由于迷宫的外周淋巴压力降低而引起的继发性感觉神经性听力损失。坐在隔音室内时，对患者的听力进行听觉控制。通过整个音阶上的空气传导来确定患者的声音感知的初始阈值。然后，患者将头部紧紧按压到胸骨，并将头部保持在该位置55-65秒。在此背景下，患者立即通过整个音阶中不同声音频率的空气传导来确定其声音感知阈值是否存在变化。另外，俯卧位的其他患者在60-65秒内连续进行深呼吸，呼吸频率为每分钟20-22。在低颅内压稳定60-65秒后，患者在整个音阶上以不同的声音频率以5 dB的步幅进行空气传导的声音感知阈值是否存在变化。如果在至少两个不同的发声频率上发现随着酒液压力的升高，通过患者的空气传导将声音感知阈值降低10 dB或更多的事实，并且同时检测到声音感知阈值的增加，在不小于两个不同的发声频率下，随着颅内压的降低，患者的空气传导增加了10 dB或更多，这被诊断为迷路的外周淋巴压力降低而导致继发性感音神经性听力丧失。通过确定当患者进行最大程度的紧缩时，通过不同声频下整个音阶上的空气传导来确定其声音感知阈值的变化，从而可以高度可靠地诊断由于淋巴周迷路压力降低而引起的继发性感音神经性听力损失头到胸骨的位置及其保留和患者的深呼吸-呼气。; 1 cl，3 ex
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