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Measuring amplifier with background adjustment and method therefor

机译：具有背景调整的测量放大器及其方法

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摘要

A measuring amplifier (103) with background calibration and adjustment amplifies, digitizes and processes at least one measurement signal (111) from at least one measuring transducer (102) with the aid of at least one amplifier arrangement (108). This can be intermittently replaced by an additional amplifier arrangement (107), which enables interruption-free direct calibration and, if necessary, adjustment of the amplifier arrangement. In the calibration, both a zero point error and an amplification error of the amplifier arrangement are reliably determined. A high accuracy is achieved without measurement interruption. Only one additional amplifier arrangement is generally required, even for a measuring amplifier with plural channels.

机译：具有背景校准和调整功能的测量放大器（ 103 ）可放大，数字化并处理来自至少一个测量换能器（ 102 < / B>）借助至少一个放大器装置（ 108 ）。可以间歇地用附加的放大器装置（ 107 ）代替，该放大器装置可实现无中断的直接校准，并在必要时进行放大器装置的调整。在校准中，可靠地确定放大器装置的零点误差和放大误差。在不中断测量的情况下实现了高精度。即使对于具有多个通道的测量放大器，通常也仅需要一个附加的放大器装置。 展开▼

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公开/公告号US10295383B2

专利类型

公开/公告日2019-05-21

原文格式PDF

申请/专利权人 HOTTINGER BALDWIN MESSTECHNIK GMBH;
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申请/专利号US201414916398

发明设计人 MARCO M. SCHAECK;HERBERT KITZING;
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申请日2014-09-05

分类号H03F3/45;H03M1/10;H03M1/12;G01D18;

国家 US

入库时间 2022-08-21 12:15:04

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1. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004）（P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期

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