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PARTICLE DETECTOR CAPABLE OF SEPARATING IN-TIME SIGNALS FROM OUT-OF-TIME SIGNALS

机译：能够将及时信号与不及时信号分离的特定检测器

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摘要

Silicon Particle Detector, comprising an absorption region (10) capable of generating electrical charges in response to a particle passing therethrough, a first and a second electrode (20, 30) arranged on opposite sides of the absorption region (10), wherein the first electrode (20) is segmented into a plurality of pads (20a), and a plurality of multiplication layers (40) able to avalanche-multiply the electric charges generated in the absorption region (10), each of the multiplication layers (40) being arranged beneath a respective pad (20a) and interposed between it and the absorption region (10), each multiplication layer (40) is surrounded by a respective protection ring (50) formed by the material of the pad (20a). The protection ring (50) is laterally interposed between the multiplication layer (40) and the absorption region (10).

机译：硅颗粒检测器，包括一个吸收区（ 10 ），该吸收区能够响应穿过的颗粒而产生电荷，并布置有第一和第二电极（ 20、30 ）在吸收区（ 10 ）的相对侧，其中第一电极（ 20 ）分为多个焊盘（ 20 a ）和多个乘法层（ 40 ），它们能够雪崩倍增吸收区（ 10 ）中产生的电荷，每个乘法层（ 40 ）布置在相应的焊盘（ 20 a ）下方，并介于其和吸收区（ 10 ），每个乘法层（ 40 ）都由由焊盘（ 20 a ）。保护环（ 50 ）横向插入在倍增层（ 40 ）和吸收区域（ 10 ）之间。 展开▼

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公开/公告号US2019198704A1

专利类型

公开/公告日2019-06-27

原文格式PDF

申请/专利权人 ISTITUTO NAZIONALE DI FISICA NUCLEARE;
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申请/专利号US201716331241

发明设计人 NICOLÒ CARTIGLIA;GIAN FRANCO DALLA BETTA;LUCIO PANCHERI;MAURIZIO BOSCARDIN;GIOVANNI PATERNOSTER;
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申请日2017-09-12

分类号H01L31/118;H01L31/028;H01L31/0224;

国家 US

入库时间 2022-08-21 12:07:41

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1. 信号稳定性检测器及时延测试装置 [P] . 中国专利： CN102221671B . 2013.04.03

2. 信号稳定性检测器及时延测试装置 [P] . 中国专利： CN102221671A . 2011-10-19

3. A PARTICLE DETECTOR CAPABLE OF SEPARATING IN-TIME SIGNALS FROM OUT-OF-TIME SIGNALS [P] . 欧洲知识产权局专利： EP3513441A1 . 2019-07-24

机译：能够将及时信号与不及时信号分离的粒子检测器

4. A PARTICLE DETECTOR CAPABLE OF SEPARATING IN-TIME SIGNALS FROM OUT-OF-TIME SIGNALS [P] . 世界知识产权组织专利： WO2018051226A1 . 2018-03-22

机译：能够将及时信号与不及时信号分离的粒子检测器

5. particle detector capable of separating signals from signals on late. [P] . IT201600092430A1 . 2018-03-14

机译：能够将信号与后期信号分离的粒子探测器。

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1. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004）（P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期

2. 杏(Prunus armeniaca L.)自交不亲和性相关S-RNase基因的检测及时空表达特性 [C] . 冯建荣 ,新疆石河子大学园艺系 ,陈学森 . 中国园艺学会第十届会员代表大会暨学术研讨会 . 2005

3. 微波光子波形信号生成及时间拉伸模数转换技术研究 [A] . 肖进勃 . 2018