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Detecting and localization method of unknown signal using aircraft with ADS-B system

机译：具有ADS-B系统的飞机对未知信号的检测与定位方法

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摘要

A method of detecting an unknown signal and estimating a source location of the unknown signal using aircraft based on an automatic dependent surveillance-broadcast (ADS-B) system is provided. The method includes a first step (S100′) for obtaining from a plurality of airborne aircrafts provided with a network system, an aircraft signal transmitted to neighboring aircraft. The method further includes a second step (S200′) for detecting, by one of the plurality of aircraft, a presence of the unknown signal in the aircraft signal based on one of a time difference of arrival (TDOA) method, a time of arrival (TOA) method, and an angle of arrival (AOA) method. The method further includes a third step (S300′) for estimating the source location of the unknown signal and a fourth step (S400′) for transmitting unknown signal generation information and the source to neighboring aircraft and the ATC through a flight information services-broadcast (FIS-B).

机译：提供了一种基于自动相关监视广播（ADS-B）系统的使用飞行器检测未知信号并估计未知信号的源位置的方法。该方法包括第一步骤（S 100 ＆＃x2032;），该步骤用于从配备有网络系统的多个空中飞机获得发送到相邻飞机的飞机信号。该方法还包括第二步骤（S 200 ＆＃x2032;），该第二步骤用于通过多个飞机之一检测基于时间差之一的飞机信号中未知信号的存在。到达时间（TDOA）方法，到达时间（TOA）方法和到达角度（AOA）方法。该方法还包括用于估计未知信号的源位置的第三步骤（S 300 ＆＃x2032;）和第四步骤（S 400 ＆＃x2032;）用于通过飞行信息服务广播（FIS-B）向邻近的飞机和ATC传输未知信号生成信息和源。 展开▼

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公开/公告号US10539679B2

专利类型

公开/公告日2020-01-21

原文格式PDF

申请/专利权人 KOREA AEROSPACE RESEARCH INSTITUTE;
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申请/专利号US201615214223

发明设计人 YOUNGSUN YUN;JEONGHO CHO;MOONBEOM HEO;GIWOOK NAM;
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申请日2016-07-19

分类号G01S19/21;G01S5/02;G01S5/06;G01S5;G01S5/04;G01S19/03;

国家 US

入库时间 2022-08-21 11:27:39

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3. Detecting and localization method of unknown signal using aircraft with ADS-B system [P] . 美国专利： US10429513B2 . 2019-10-01

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机译：具有ADS-B系统的飞机对未知信号的检测与定位方法

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1. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004）（P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期

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