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Reflex epilepsy and non-ketotic hyperglycemia.

机译:反射性癫痫和非酮症高血糖症。

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摘要

Epileptic seizures are paroxysmal events, and it is likely that many, if not most, of them are precipitated by known or unknown factors acting on a central nervous system that is predisposed to the production of epileptic discharges by the presence of an organic lesion, a genetically determined neuronal hyperexcitability, or both. Known precipitating influences are quite varied. We report the case of a 58 years-old patient, followed for non-ketotic hyperglycemia, who presented with focal seizures exclusively induced by specific, active or passive, postures and movements of the right arm or hand. MRI was normal. Following regulation of glucose blood levels the position-induced seizures stopped and antiepileptics were not prescribed. The seizures are kept under control by regulating blood glucose.
机译:癫痫性发作是阵发性事件,很可能许多(如果不是大多数)是由作用于中枢神经系统的已知或未知因素引起的,中枢神经系统易因存在有机性病变,基因确定的神经元过度兴奋性,或两者兼而有之。已知的沉淀影响差异很大。我们报道了一例58岁的患者,其次是非酮症性高血糖症,其表现为仅由右臂或手的特定,主动或被动,姿势和运动引起的局灶性癫痫发作。 MRI正常。在调节血糖水平后,停止了由位置引起的癫痫发作,并且未开具抗癫痫药。通过调节血糖控制癫痫发作。

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