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Multiprotocol-induced plasticity in artificial synapses

机译:Multiprotocol-induced可塑性在人工突触

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摘要

We suggest a 'universal' electrical circuit for the realization of an artificial synapse that exhibits long-term plasticity induced by different protocols. The long-term plasticity of the artificial synapse is basically attributed to the nonvolatile resistance change of the bipolar resistive switch in the circuit. The synaptic behaviour realized by the circuit is termed 'universal' inasmuch as (i) the shape of the action potential is not required to vary so as to implement different plasticity-induction behaviours, activity-dependent plasticity (ADP) and spike-timing-dependent plasticity (STDP), (ii) the behaviours satisfy several essential features of a biological chemical synapse including firing-rate and spike-timing encoding and unidirectional synaptic transmission, and (iii) both excitatory and inhibitory synapses can be realized using the same circuit but different diode polarity in the circuit. The feasibility of the suggested circuit as an artificial synapse is demonstrated by conducting circuit calculations and the calculation results are introduced in comparison with biological chemical synapses.
机译:我们建议一个“通用”电路实现一个人工突触展品长期引起的可塑性不同的协议。人工突触基本上是归因于双相的非易失性阻力变化电阻电路的开关。行为实现的电路称为“通用”因为(我)的形状动作电位不需要以不同实现不同plasticity-induction行为,活动依赖性可塑性(ADP)和spike-timing-dependent可塑性(STDP),(2)行为满足几个重要生物化学突触的特性包括燃烧率和spike-timing编码和单向突触传递(3)兴奋性和抑制性突触使用相同的电路实现,但不同电路中二极管极性。显示电路作为人工突触证明了进行电路计算并介绍了计算结果与生物化学突触。

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