机译:CD56明亮的天然杀手细胞通过细胞分解和CCCDNA衰减在长期Entecavir治疗的患者切换到Peginterferon Alfa-2a的患者中诱导HBsAg降低
Department and Institute of Infectious DiseaseHuazhong University of Science and TechnologyWuhan;
Department and Institute of Infectious DiseaseHuazhong University of Science and TechnologyWuhan;
Department and Institute of Infectious DiseaseHuazhong University of Science and TechnologyWuhan;
Department and Institute of Infectious DiseaseHuazhong University of Science and TechnologyWuhan;
Department and Institute of Infectious DiseaseHuazhong University of Science and TechnologyWuhan;
Department and Institute of Infectious DiseaseHuazhong University of Science and TechnologyWuhan;
Department of Pediatric DiseaseHuazhong University of Science and TechnologyWuhan China;
Department and Institute of Infectious DiseaseHuazhong University of Science and TechnologyWuhan;
Department and Institute of Infectious DiseaseHuazhong University of Science and TechnologyWuhan;
chronic hepatitis B; covalently closed circular DNA; hepatitis B surface antigen; natural killer cell; peginterferon alfa‐2a;
机译:CD56明亮的天然杀手细胞通过细胞分解和CCCDNA衰减在长期Entecavir治疗的患者切换到Peginterferon Alfa-2a的患者中诱导HBsAg降低
机译:CD56明亮的天然杀手细胞通过细胞分解和CCCDNA衰减在长期Entecavir治疗的患者切换到Peginterferon Alfa-2a的患者中诱导HBsAg降低
机译:PEG-Interferonα诱导CD56(明亮)天然杀手细胞介导的恩替卡韦治疗慢性乙型肝炎患者HBsAg的减少
机译:更正:具有重组酶激活基因和非同源末端连接基因缺陷的患者的自然杀伤细胞包含较高频率的CD56bright NKG2A +++细胞但显示出增加的去颗粒和较高的穿孔素含量
机译:更正:来自重组酶激活基因和非同源末端连接基因缺陷患者的自然杀伤细胞包含较高频率的CD56(亮)NKG2A(+++)细胞,但显示出增加的去颗粒化和较高的穿孔素含量