机译:Wisp1通过在骨关节炎中调节αVβ3和PI3K / AKT途径来保护软骨细胞衰老和细胞凋亡
Yiyang Cent Hosp Dept Orthopaed Yiyang 413000 Peoples R China;
Cent South Univ Xiangya Hosp Dept Orthopaed Changsha 410008 Peoples R China;
Cent South Univ Xiangya Hosp Dept Emergency Changsha Peoples R China;
Shenzhen Univ Shenzhen Peoples Hosp 2 Affiliated Hosp 1 Dept Sports Med Shenzhen Peoples R China;
Second Peoples Hosp Jingmen Dept Orthoped Jingmen Peoples R China;
Changsha Cent Hosp Dept Orthoped Changsha Peoples R China;
Yiyang Cent Hosp Dept Orthopaed Yiyang 413000 Peoples R China;
Yiyang Cent Hosp Dept Orthopaed Yiyang 413000 Peoples R China;
Yiyang Cent Hosp Dept Orthopaed Yiyang 413000 Peoples R China;
Yiyang Cent Hosp Dept Orthopaed Yiyang 413000 Peoples R China;
Cent South Univ Xiangya Hosp Dept Orthopaed Changsha 410008 Peoples R China;
Yiyang Cent Hosp Dept Orthopaed Yiyang 413000 Peoples R China;
osteoarthritis; WISP1; chondrocyte; senescence; apoptosis;
机译:MiR-34a通过靶向DLL1和调节PI3K / AKT途径增强软骨细胞的凋亡,衰老并促进骨关节炎的发展。
机译:FAM3A通过调节PI3K / AKT / MTOR途径来保护软骨细胞免受白细胞介素-1β诱导的细胞凋亡
机译:SIRT3通过PI3K / AKT / MTOR途径调节软骨细胞自噬和细胞凋亡来改善骨关节炎
机译:使用LTQ orbitrap MS / MS分析确定通过PI3K / AKT信号通路凋亡中角蛋白长丝在细胞凋亡中的功能作用
机译:DLX4同源蛋白通过上调VEGFA促进ER阴性乳腺癌细胞中的PI3K / Akt抗凋亡途径。
机译:Shikonin通过调节PI3K / Akt信号通路保护软骨细胞免受白介素-1β诱导的凋亡
机译:MiR-34A通过靶向DLL1和调节PI3K / AKT路径来增强软骨细胞凋亡,衰老,促进骨关节炎的发育
机译:分子靶向pI3K / akt通路预防激素抵抗前列腺癌的发生