机译:SIRT3通过PI3K / AKT / MTOR途径调节软骨细胞自噬和细胞凋亡来改善骨关节炎
Zhejiang Univ Affiliated Hosp 2 Dept Orthoped Surg Sch Med Hangzhou Peoples R China;
Zhejiang Univ Affiliated Hosp 2 Dept Orthoped Surg Sch Med Hangzhou Peoples R China;
Zhejiang Univ Affiliated Hosp 2 Dept Orthoped Surg Sch Med Hangzhou Peoples R China;
Zhejiang Univ Affiliated Hosp 2 Dept Orthoped Surg Sch Med Hangzhou Peoples R China;
Zhejiang Univ Affiliated Hosp 2 Dept Orthoped Surg Sch Med Hangzhou Peoples R China;
Zhejiang Univ Affiliated Hosp 2 Dept Orthoped Surg Sch Med Hangzhou Peoples R China;
SIRT3; Osteoarthritis; Autophagy; Apoptosis; PI3K/Akt/mTOR signaling;
机译:MiR-4262通过靶向SIRT1和激活骨关节炎大鼠的PI3K / AKT / MTOR信号传导途径来调节软骨细胞活力,细胞凋亡,自噬
机译:β-Ecdysterone通过调节PI3K / AKT / MTOR信号途径激活软骨细胞中的自噬,减轻骨关节炎
机译:艺术术对软骨细胞增殖,细胞凋亡和自噬的影响,具有类风湿性关节炎大鼠模型中的PI3K / AKT / MTOR信号通路
机译:阳离子聚苯乙烯纳米球通过抑制Akt / mTOR和激活巨噬细胞和上皮细胞中的AMPK信号通路来诱导自噬
机译:PI3k / AKT / mTOR信号通路的遗传分析。
机译:miR-4262通过靶向SIRT1并激活骨关节炎大鼠的PI3K / AKT / mTOR信号通路来调节软骨细胞的活力凋亡自噬
机译:MiR-27A通过PI3K / AKT / MTOR信号传导促进骨关节炎IL-1β处理关节细胞细胞的自噬和凋亡
机译:pI3K / akt / mTOR网络中网络状态和药物反应的实验分析和计算模型