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机译:miR-322 / miR-503簇通过靶向celf1调节肌细胞营养不良RNA毒性的缺陷肌细胞分化
Nanchang Univ Dept Cardiovasc Affiliated Hosp 1 17 Yongwai Zheng St Nanchang 330006 Jiangxi Peoples R China;
Nanchang Univ Dept Cardiovasc Affiliated Hosp 1 17 Yongwai Zheng St Nanchang 330006 Jiangxi Peoples R China;
Nanchang Univ Dept Cardiovasc Affiliated Hosp 1 17 Yongwai Zheng St Nanchang 330006 Jiangxi Peoples R China;
Nanchang Univ Dept Cardiovasc Affiliated Hosp 1 17 Yongwai Zheng St Nanchang 330006 Jiangxi Peoples R China;
Nanchang Univ Dept Cardiovasc Affiliated Hosp 1 17 Yongwai Zheng St Nanchang 330006 Jiangxi Peoples R China;
Nanchang Univ Dept Cardiovasc Affiliated Hosp 1 17 Yongwai Zheng St Nanchang 330006 Jiangxi Peoples R China;
Nanchang Univ Dept Cardiovasc Affiliated Hosp 1 17 Yongwai Zheng St Nanchang 330006 Jiangxi Peoples R China;
Nanchang Univ Dept Cardiovasc Affiliated Hosp 1 17 Yongwai Zheng St Nanchang 330006 Jiangxi Peoples R China;
miR-322; miR-503; Celf1; myotonic dystrophy; myoblast differentiation;
机译:Celf1调节细胞周期,部分导致肌强直性营养不良RNA毒性中成肌细胞分化不良
机译:MiR-322 / -503通过靶向凸型重复来拯救肌细胞缺陷型细胞模型中的肌细胞缺陷
机译:肌强直性营养不良蛋白激酶(DMPK)的水平降低和人类强直性肌营养不良成肌细胞的分化延迟。
机译:强直性肌营养不良症突变对肌肉分化和凋亡的影响。
机译:MiR-322 / -503通过靶向凸型重复来拯救肌细胞缺陷型细胞模型中的肌细胞缺陷
机译:Celf1调节细胞周期,部分导致肌强直性营养不良RNA毒性中成肌细胞分化不良