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机译:通过累积ROS来抑制PI3K / AKT / MTOR信号通路,eldecalcitol诱导人骨瘤瘤Mg-63细胞中的凋亡和自噬
Shandong Univ Shandong Key Lab Oral Tissue Regenerat Shandong Engn Lab Dent Mat &
Oral Tissue Regenera Dept Bone Metab Sch &
Hosp Stomatol Cheeloo Coll Jinan 250012 Peoples R China;
Shandong Univ Shandong Key Lab Oral Tissue Regenerat Shandong Engn Lab Dent Mat &
Oral Tissue Regenera Dept Bone Metab Sch &
Hosp Stomatol Cheeloo Coll Jinan 250012 Peoples R China;
Shandong Univ Shandong Key Lab Oral Tissue Regenerat Shandong Engn Lab Dent Mat &
Oral Tissue Regenera Dept Bone Metab Sch &
Hosp Stomatol Cheeloo Coll Jinan 250012 Peoples R China;
Shandong Univ Shandong Key Lab Oral Tissue Regenerat Shandong Engn Lab Dent Mat &
Oral Tissue Regenera Dept Bone Metab Sch &
Hosp Stomatol Cheeloo Coll Jinan 250012 Peoples R China;
Shandong Univ Shandong Key Lab Oral Tissue Regenerat Shandong Engn Lab Dent Mat &
Oral Tissue Regenera Dept Bone Metab Sch &
Hosp Stomatol Cheeloo Coll Jinan 250012 Peoples R China;
Eldecalcitol; Apoptosis; Autophagy; Osteosarcoma; ROS; PI3K/Akt/mTOR pathway;
机译:二烯丙基二硫键通过PI3K / Akt / mTOR途径诱导人骨肉瘤MG-63细胞凋亡和自噬
机译:组织蛋白酶S的抑制通过ROS介导的PI3K / AKT / mTOR / p70S6K和JNK信号通路诱导人胶质母细胞瘤细胞系自噬和凋亡
机译:类黄酮通过下调PI3Kγ介导的PI3K / AKT / mTOR / p70S6K / ULK信号通路抑制人乳腺癌细胞的增殖并诱导细胞凋亡和自噬
机译:阳离子聚苯乙烯纳米球通过抑制Akt / mTOR和激活巨噬细胞和上皮细胞中的AMPK信号通路来诱导自噬
机译:多靶点受体酪氨酸激酶抑制剂利尼法尼(ABT-869)通过磷酸肌醇3激酶(PI3K)/ AKT依赖性信号传导途径诱导凋亡并抑制白血病细胞的增殖。
机译:二烯丙基二硫键通过PI3K / Akt / mTOR途径诱导人骨肉瘤MG-63细胞凋亡和自噬
机译:Danusertib诱导细胞凋亡,细胞周期阻滞和自噬,但抑制上皮细胞向间质转化,涉及pI3K / akt / mTOR信号通路在人类卵巢癌细胞中的表达
机译:暴露应用Fas受体小干扰RNa抑制硫芥诱导人气道上皮细胞凋亡:治疗方法的意义。