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机译:抑制蛋白酶体B2位点对B5抑制剂的三阴性乳腺癌细胞感染并抑制NRF1活化
Department of Pharmacology and Toxicology Geisel School of Medicine at Dartmouth Lebanon NH 03756 USA;
Department of Pharmacology and Toxicology Geisel School of Medicine at Dartmouth Lebanon NH 03756 USA;
Department of Cell Biology Harvard Medical School Boston MA 02115 USA;
Department of Pharmacology and Toxicology Geisel School of Medicine at Dartmouth Lebanon NH 03756 USA;
Department of Chemistry Dartmouth College Hanover NH 03755 USA;
Gorlaeus Laboratories Leiden Institute of Chemistry 2333 CC Leiden the Netherlands;
Gorlaeus Laboratories Leiden Institute of Chemistry 2333 CC Leiden the Netherlands;
Department of Cell Biology Harvard Medical School Boston MA 02115 USA;
Department of Pharmacology and Toxicology Geisel School of Medicine at Dartmouth Lebanon NH 03756 USA;
Department of Pharmacology and Toxicology Geisel School of Medicine at Dartmouth Lebanon NH 03756 USA;
Inhibition of Proteasome; Sensitizes Triple-Negative; Nrf1 Activation;
机译:拉帕替尼诱导的NF-κB活化使三阴性乳腺癌细胞对蛋白酶体抑制剂敏感
机译:Rad51抑制使乳腺癌干细胞对三阴性乳腺癌中的PARP抑制剂敏感
机译:双靶MDM2 / MDMX抑制剂增加了多柔比星的敏化,并通过激活Tab1 / Tak1 / P38 Mapk路径来抑制三阴性乳腺癌细胞的迁移和侵袭能力
机译:纳米颗粒涂有Frizzled7抗体使三价乳腺癌中增强Wnt信号抑制的多价结合。
机译:ErbB-2的抑制通过Notch-1及其配体JAG-1的细胞表面积聚来激活Notch-1,并使ErbB-2阳性乳腺癌细胞对GSI敏感
机译:蛋白酶体β2位点的抑制通过涉及Nrf1抑制的机制使三阴性乳腺癌细胞对β5抑制剂敏感
机译:抑制蛋白酶体β2位点对β5抑制剂的三阴性乳腺癌细胞感染并抑制NRF1活化
机译:靶向三阴性乳腺癌中的乳腺癌干细胞。