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机译:褪黑激素通过AKT / NRF2 / GPX4信号通路改善缺氧缺血性脑损伤
Chengdu Med Coll Affiliated Hosp 1 Dept Pediat Chengdu 610500 Sichuan Peoples R China;
Sichuan Univ West China Univ Hosp 2 Dept Pediat Minist Educ Key Lab Birth Defects &
Related Dis;
Chengdu Med Coll Affiliated Hosp 1 Dept Pediat Chengdu 610500 Sichuan Peoples R China;
Chengdu Med Coll Affiliated Hosp 1 Dept Pediat Chengdu 610500 Sichuan Peoples R China;
Chengdu Med Coll Affiliated Hosp 1 Dept Pediat Chengdu 610500 Sichuan Peoples R China;
Chengdu Med Coll Affiliated Hosp 1 Dept Pediat Chengdu 610500 Sichuan Peoples R China;
Chengdu Med Coll Affiliated Hosp 1 Clin Lab Chengdu 610500 Sichuan Peoples R China;
Chengdu Med Coll Affiliated Hosp 1 Dept Pediat Chengdu 610500 Sichuan Peoples R China;
Melatonin; Hypoxic-ischemic brain damage; Neuron; Ferroptosis; Akt/Nrf2/Gpx4;
机译:N-乙酰胆管素衍生物通过抑制NLRP3炎性组织并在缺氧缺血性脑损伤模型中激活PI3K / AKT / NRF2途径来施加神经保护作用
机译:通过PI3K / AKT / NRF2信号传导途径激活新生大鼠肝蛋白对新生大鼠缺氧缺血性脑损伤的神经保护作用
机译:姜黄素靶向血管内皮生长因子,通过激活PI3K / AKT信号通路,提高新生大鼠的脑缺氧缺血性损伤
机译:新生大鼠缺氧缺血性脑损伤与心跳间隔信息的关系
机译:与胃平滑肌中褪黑激素受体MT1偶联的信号通路。
机译:姜黄素靶向血管内皮生长因子通过激活PI3K / AKT信号通路提高新生大鼠的脑缺氧缺血性损伤
机译:冬青菌素通过减轻氧化应激和NRF2 / HO-1 /是信号通路,衰减新生大鼠的缺氧缺血性脑损伤
机译:在乳腺癌进展中通过pI 3-K,akt和sGK途径发出信号。