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机译:丧失选择性自噬受体P62损害小鼠髓性白血病进展和水肿
Goethe Univ Dept Med Hematol Oncol Frankfurt Germany;
Goethe Univ Dept Med Hematol Oncol Frankfurt Germany;
Goethe Univ Dept Med Hematol Oncol Frankfurt Germany;
Goethe Univ Dept Med Hematol Oncol Frankfurt Germany;
Goethe Univ Inst Biochem 2 Frankfurt Germany;
Goethe Univ Dept Med Hematol Oncol Frankfurt Germany;
Goethe Univ Dept Med Hematol Oncol Frankfurt Germany;
Goethe Univ Dept Med Hematol Oncol Frankfurt Germany;
Goethe Univ Dept Med Hematol Oncol Frankfurt Germany;
Goethe Univ Dept Med Hematol Oncol Frankfurt Germany;
Goethe Univ Inst Cardiovasc Regenerat Frankfurt Germany;
Univ Hosp Heidelberg Dept Hematol Oncol &
Rheumatol Heidelberg Germany;
Goethe Univ Inst Biol &
Neurosci Frankfurt Germany;
Goethe Univ Inst Mol Biosci Frankfurt Germany;
Goethe Univ Inst Biochem 2 Frankfurt Germany;
Goethe Univ Inst Biochem 2 Frankfurt Germany;
German Canc Consortium Heidelberg Germany;
Goethe Univ Dept Med Hematol Oncol Frankfurt Germany;
机译:选择性自噬受体的丧失P62损失小鼠髓性白血病进展和MINOCHAGY(第133号,PG 168,2019)
机译:丧失选择性自噬受体p62延迟白血病发展和损害斑驳
机译:丧失选择性自噬受体p62延迟白血病发展和损害斑驳
机译:正常髓样发育中菌落刺激因子3受体(CSF3R)的磷脂蛋白酶的扰动,髓性白血病和中性白血病
机译:Egr1的丢失在小鼠白血病的发生中起作用,并且可能在急性髓细胞性白血病和与治疗有关的急性髓细胞性白血病的发展中起作用。
机译:p62 / sequestosome1的泛素化激活其自噬受体功能并在泛素应激时控制选择性自噬
机译:Nguyen TD,Shait S,Vakhrusheva O,等。丧失选择性自噬受体P62损害小鼠髓性白血病进展和水肿。血液。 2019; 133(2):168-179。