机译:选择性HDL培养人APO A-I基因治疗抵消了心肌肥大,减少了心肌纤维化,并改善了慢性压力过载的小鼠中的心脏功能
Katholieke Univ Leuven Dept Cardiovasc Sci Ctr Mol &
Vasc Biol B-3000 Leuven Belgium;
Katholieke Univ Leuven Dept Cardiovasc Sci Ctr Mol &
Vasc Biol B-3000 Leuven Belgium;
Katholieke Univ Leuven Dept Cardiovasc Sci Ctr Mol &
Vasc Biol B-3000 Leuven Belgium;
Katholieke Univ Leuven Dept Cardiovasc Sci Ctr Mol &
Vasc Biol B-3000 Leuven Belgium;
Katholieke Univ Leuven Dept Cardiovasc Sci Ctr Mol &
Vasc Biol B-3000 Leuven Belgium;
Katholieke Univ Leuven Dept Cardiovasc Sci Ctr Mol &
Vasc Biol B-3000 Leuven Belgium;
high-density lipoproteins; gene therapy; cardiac hypertrophy; heart failure; pressure overload; cardiac function; oxidative stress; apolipoprotein A-I;
机译:选择性HDL升高人类Apo A-I基因治疗可抵消心脏肥大,减少心肌纤维化并改善患有慢性压力超负荷的小鼠的心脏功能
机译:选择性HDL升高基因转移对小鼠心肌梗死后存活,心脏重塑和心功能的有益作用
机译:Tenascin-C促进慢性压力过载诱导的心脏功能障碍,肥大和心肌纤维化
机译:药物和细胞疗法可减少急性心肌梗死后的病理重塑和心脏功能障碍
机译:选择性HDL升高人类Apo A-I基因治疗可抵消心脏肥大减少心肌纤维化并改善患有慢性压力超负荷的小鼠的心脏功能
机译:选择性HDL升高人类Apo A-I基因治疗可抵消心脏肥大,减少心肌纤维化并改善患有慢性压力超负荷的小鼠的心脏功能
机译:红细胞储存对心脏功能的影响。常温和低温下低氧血红蛋白亲和力人红细胞恒流冠状动脉灌注时心肌氧输送和功能的改善