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机译:SeNP2通过SetdB1的De-Suflation调节脂肪脂质储存
Shanghai Jiao Tong Univ Sch Med Shanghai Key Lab Tumor Microenvironm &
Inflammat Dept Biochem &
Shanghai Jiao Tong Univ Sch Med Shanghai Key Lab Tumor Microenvironm &
Inflammat Dept Biochem &
Shanghai Jiao Tong Univ Sch Med Shanghai Key Lab Tumor Microenvironm &
Inflammat Dept Biochem &
Shanghai Jiao Tong Univ Ruijin Hosp Dept Endocrinol &
Metab Sch Med Shanghai Peoples R China;
Shanghai Jiao Tong Univ Sch Med Shanghai Key Lab Tumor Microenvironm &
Inflammat Dept Biochem &
Shanghai Jiao Tong Univ Sch Med Shanghai Key Lab Tumor Microenvironm &
Inflammat Dept Biochem &
Fudan Univ Shanghai Med Coll Inst Biomed Sci Canc Metab Lab Shanghai 200032 Peoples R China;
Shanghai Jiao Tong Univ Sch Med Shanghai Key Lab Tumor Microenvironm &
Inflammat Dept Biochem &
Shanghai Jiao Tong Univ Sch Med Shanghai Key Lab Tumor Microenvironm &
Inflammat Dept Biochem &
Shanghai Jiao Tong Univ Sch Med Shanghai Key Lab Tumor Microenvironm &
Inflammat Dept Biochem &
Shanghai Jiao Tong Univ Sch Med Shanghai Key Lab Tumor Microenvironm &
Inflammat Dept Biochem &
lipid storage; Senp2; Setdb1; H3K9me3; Pparg and Cebpa;
机译:Senp2通过SetDB1的脱SUMOylation调节脂肪脂质的存储
机译:Senp2通过SetDB1的脱SUMOylation调节脂肪脂质的存储
机译:Senp2通过Setdbl的脱SUMOylation调节脂肪脂质的存储
机译:人的移植脂肪组织衍生的多谱系祖细胞但不是脂肪组织衍生的基质/干细胞减少高脂血症Watanabe兔中的血清胆固醇
机译:肝脏,脂肪和肌肉的脂质代谢和糖皮质激素的肌肉和这些器官如何互相调节
机译:Sentrin / Sumo特异性蛋白酶2(SeNP2)的去雄性酶调节干扰诱导的ERK5和P53的流动诱导的雄性导致内皮功能障碍和动脉粥样硬化
机译:SENP2通过SETDB1的DE-SUMLATION调节脂肪脂质储存