...
机译:长期非编码RNA MALAT1通过冲把MIR-101促进通过放弃RAP1B来抑制胶质瘤细胞的凋亡并抑制胶质瘤细胞的凋亡
Henan Univ Dept Neurosurg Huaihe Hosp 1 Baogonghu North Rd Gulou Dist 475000 Kaifeng Peoples;
Henan Univ Dept Neurosurg Huaihe Hosp 1 Baogonghu North Rd Gulou Dist 475000 Kaifeng Peoples;
Henan Univ Dept Neurosurg Huaihe Hosp 1 Baogonghu North Rd Gulou Dist 475000 Kaifeng Peoples;
Henan Univ Dept Neurosurg Huaihe Hosp 1 Baogonghu North Rd Gulou Dist 475000 Kaifeng Peoples;
Zhengzhou Univ Dept Neurosurg Affiliated Hosp 1 Zhengzhou 450003 Henan Peoples R China;
Capital Med Univ Beijing Tian Tan Hosp Dept Neurosurg Beijing 100071 Peoples R China;
MALAT1; MiR; 101; Rap1B; Proliferation; Apoptosis; Glioma;
机译:长期非编码RNA MALAT1通过冲把MIR-101促进通过放弃RAP1B来抑制胶质瘤细胞的凋亡并抑制胶质瘤细胞的凋亡
机译:MALAT1促进MiR-214调节TRPV4表达,从而促进细胞凋亡并抑制睾丸缺血再灌注损伤中的细胞增殖。
机译:长期非编码RNA MALAT1通过海绵MICRORNA-146A调节急性髓性白血病中的CXCR4表达调节细胞迁移,增殖和凋亡
机译:长非编码RNA MALAT1和MENβ/ε的进化
机译:Neurexophilin1抑制造血祖细胞的增殖
机译:长的非编码RNA Malat1激活自噬因此通过在结肠直肠癌中掺入miR-101来促进细胞增殖并抑制细胞凋亡
机译:Malat1促进细胞凋亡并通过冲水MIR-214调节TRPV4表达,抑制睾丸缺血再灌注损伤中的细胞增殖