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Ocular Effects of Exposure to 40, 75, and 95 GHz Millimeter Waves

机译:暴露于40,75和95GHz毫米波的眼睛效应

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摘要

The objective of this study was to develop a model of ocular damage induced by 40, 75, and 95 GHz continuous millimeter waves (MMW), thereby allowing assessment of the clinical course of ocular damage resulting from exposure to thermal damage-inducing MMW. This study also examined the dependence of ocular damage on incident power density. Pigmented rabbit eyes were exposed to 40, 75, and 95 GHz MMW from a spot-focus-type lens antenna. Slight ocular damage was observed 10 min after MMW exposure, including reduced cornea thickness and reduced transparency. Diffuse fluorescein staining around the pupillary area indicated corneal epithelial injury. Slit-lamp examination 1 day after MMW exposure revealed a round area of opacity, accompanied by fluorescence staining, in the central pupillary zone. Corneal edema, indicative of corneal stromal damage, peaked 1 day after MMW exposure, with thickness gradually subsiding to normal. Three days after exposure, ocular conditions had almost normalized, though corneal thickness was slightly greater than that before exposure. The 50% probability of ocular damage (DD50) was in the order 40 95 ae 75 GHz at the same incident power densities.
机译:本研究的目的是开发由40,75和95GHz连续毫米波(MMW)引起的眼部损伤模型,从而允许评估由暴露于热损伤诱导MMW引起的眼睛损伤的临床过程。本研究还研究了眼睛损伤对入射功率密度的依赖性。从焦点焦点型透镜天线暴露于40,75和95GHz MMW的着色兔眼。 MMW暴露后10分钟观察到轻微的眼睛损伤,包括降低角膜厚度和降低的透明度。弥漫性荧光素染色瞳孔区域表示角膜上皮损伤。 MMW暴露后1天的狭缝灯检查显示圆形不透明度,伴随着中央瞳孔区的荧光染色。角膜水肿,指示角膜基质损伤,达到MMW暴露后的1天达到峰值,厚度逐渐消退到正常。暴露后三天,眼部条件几乎归一化,但角膜厚度略大于暴露前的略高。 50%的眼镜损伤(DD50)的概率为40& 95 AE 75 GHz在相同的事件电力密度。

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