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机译:催产素衰减社会互动的缺陷,但在产前丙戊酸诱导的自闭症小鼠模型中没有识别记忆
Laboratory of Molecular Neuropharmacology Graduate School of Pharmaceutical Sciences Osaka;
Laboratory of Molecular Neuropharmacology Graduate School of Pharmaceutical Sciences Osaka;
Laboratory of Molecular Neuropharmacology Graduate School of Pharmaceutical Sciences Osaka;
Department of Pharmacology Graduate School of Dentistry Osaka University;
Laboratory of Molecular Neuropharmacology Graduate School of Pharmaceutical Sciences Osaka;
Laboratory of Molecular Neuropharmacology Graduate School of Pharmaceutical Sciences Osaka;
Laboratory of Medicinal Pharmacology Graduate School of Pharmaceutical Sciences Osaka University;
Department of Pharmacology Graduate School of Dentistry Osaka University;
Autism mouse model; Valproic acid; Oxytocin; Social interaction; Recognition memory;
机译:催产素衰减社会互动的缺陷,但在产前丙戊酸诱导的自闭症小鼠模型中没有识别记忆
机译:MS-11,NRSF中MSIN3结合螺旋的模拟物,改善了产前丙戊酸诱导的自闭症小鼠模型中的社会相互作用缺陷
机译:组胺H3R拮抗剂DL77衰减产前丙戊酸诱导的自闭症小鼠模型中的自闭症行为
机译:小鼠社交行为的无监督分析揭示了活动过度如何影响自闭症小鼠模型中的社交方面
机译:在Balb / C小鼠中,社会奖励完整,受到后丙戊酸治疗的衰减,并在自闭症的神经发育动物模型中不受GSTM1敲除的影响
机译:组胺H3R拮抗剂DL77减弱了在产前丙戊酸诱发的自闭症小鼠模型中的自闭行为
机译:催产素分化作用根据给药途径在产前丙戊酸诱导的自闭症大鼠模型中