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机译:空气污染与Apoe( - / - )小鼠的CD36和NLRP3炎症的合作开发动脉粥样硬化有关
Fudan Univ Sch Publ Hlth Dept Environm Hlth Shanghai Peoples R China;
Fudan Univ Sch Publ Hlth Dept Environm Hlth Shanghai Peoples R China;
Fudan Univ Sch Publ Hlth Dept Environm Hlth Shanghai Peoples R China;
Fudan Univ Sch Publ Hlth Dept Environm Hlth Shanghai Peoples R China;
Fudan Univ Sch Publ Hlth Dept Environm Hlth Shanghai Peoples R China;
Shanghai Key Lab Meteorol &
Hlth Shanghai Peoples R China;
Shanghai Jiao Tong Univ Sch Med Xinhua Hosp Dept Cardiol Shanghai 200092 Peoples R China;
Fudan Univ Sch Publ Hlth Dept Environm Hlth Shanghai Peoples R China;
Fudan Univ Sch Publ Hlth Dept Environm Hlth Shanghai Peoples R China;
Ohio State Univ Div Environm Hlth Sci Coll Publ Hlth Columbus OH 43210 USA;
Fudan Univ Sch Publ Hlth Dept Environm Hlth Shanghai Peoples R China;
Fine particulate matter; Atherosclerosis; NLRP3 inflammasome;
机译:空气污染与Apoe( - / - )小鼠的CD36和NLRP3炎症的合作开发动脉粥样硬化有关
机译:NLRP3炎性体的激活导致apoE缺陷小鼠高同型半胱氨酸血症加重炎症和动脉粥样硬化
机译:ApoE缺陷小鼠的动脉粥样硬化独立于NLRP3炎症小体而发展
机译:动脉粥样硬化的实验模型的作用:Apoe-Nockout小鼠在制育新药物对动脉粥样硬化的作用
机译:apoE-/-小鼠吸入环境颗粒物的半挥发性有机成分加剧动脉粥样硬化的机制。
机译:ApoE缺陷小鼠的动脉粥样硬化独立于NLRP3炎症小体而发展
机译:ApoE缺陷小鼠的动脉粥样硬化独立于NLRP3炎症小体而发展