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机译:133人组织Kallikrein 1通过Cox-2 / PTGIS / CAMP和DDAH / NOS / CGMP途径保护老年转基因大鼠的勃起功能
Tongji Hospital Tongji Medical College Huazhong University of Science and Technology;
Tongji Hospital Tongji Medical College Huazhong University of Science and Technology;
Tongji Hospital Tongji Medical College Huazhong University of Science and Technology;
Tongji Hospital Tongji Medical College Huazhong University of Science and Technology;
Tongji Hospital Tongji Medical College Huazhong University of Science and Technology;
Tongji Hospital Tongji Medical College Huazhong University of Science and Technology;
Tongji Hospital Tongji Medical College Huazhong University of Science and Technology;
Tongji Hospital Tongji Medical College Huazhong University of Science and Technology;
机译:133人组织Kallikrein 1通过Cox-2 / PTGIS / CAMP和DDAH / NOS / CGMP途径保护老年转基因大鼠的勃起功能
机译:通过抑制携带人组织激肽释放酶1的老年转基因大鼠的Rho激酶/ LIM激酶/ cofilin途径,减少体质纤维化以保护勃起功能
机译:132减少体纤维化以通过抑制患者携带人体组织Kallikrein的老年转基因大鼠中的Smad和非Smad途径来保护勃起功能
机译:在1.5 T的功能磁共振图像中从活化的脑组织中分离静脉
机译:通过战略诱变对人体组织型纤溶酶原激活物和纤溶酶原的赖氨酸结合kringle 2域的结构功能分析。
机译:DDAH / ADMA / NOS / cGMP和COX-2 / PTGIS / cAMP途径参与人激肽释放酶1保护老年大鼠勃起功能的过程
机译:DDaH / aDma / NOs / cGmp和COX-2 / pTGIs / camp通路参与人组织激肽释放酶1对老年大鼠勃起功能的保护作用。