机译:IARS2的敲低通过调节P53 / P21 / PCNA / EIF4E途径抑制急性髓性白血病细胞的增殖
Shaanxi Univ Chinese Med Affiliated Hosp 2 Weiyang West Rd Xianyang Peoples R China;
Shaanxi Univ Chinese Med Affiliated Hosp 2 Weiyang West Rd Xianyang Peoples R China;
Shaanxi Univ Chinese Med Affiliated Hosp 2 Weiyang West Rd Xianyang Peoples R China;
Shaanxi Univ Chinese Med Coll Pharm Xianyang Peoples R China;
Shaanxi Univ Chinese Med Affiliated Hosp 2 Weiyang West Rd Xianyang Peoples R China;
Shaanxi Univ Chinese Med Affiliated Hosp 2 Weiyang West Rd Xianyang Peoples R China;
Shaanxi Univ Chinese Med Affiliated Hosp 2 Weiyang West Rd Xianyang Peoples R China;
Shaanxi Univ Chinese Med Affiliated Hosp 2 Weiyang West Rd Xianyang Peoples R China;
Acute myeloid leukemia (AML); IARS2; Proliferation;
机译:IARS2的敲低通过调节P53 / P21 / PCNA / EIF4E途径抑制急性髓性白血病细胞的增殖
机译:PPM1D敲低抑制细胞增殖,促进细胞凋亡,并在急性髓性白血病中激活P38 MAPK / P53信号通路
机译:MiR-125a通过靶向急性髓性白血病中的ErbB途径调节细胞周期,增殖和凋亡
机译:消化脱脂乳对人急性髓性白血病(HL-60)细胞增殖活性的影响
机译:阐明XIAP在调节急性髓性白血病细胞死亡信号通路中的作用
机译:苦参碱通过p53 / p21 / PCNA / eIF4E信号通路减少A549细胞的增殖
机译:真核引发因子-2,γ亚基,通过MAPK / ERK信号通路在急性髓性白血病中抑制增殖并调节细胞周期